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10.1016/j.cell.2021.03.005 http://scihub22266oqcxt.onion/10.1016/j.cell.2021.03.005 33811810!8052295!33811810     free     free     free       Cell 2021 ; 184 (8): 1990-2019 Nephropedia Template TP {{tp|p=33811810|t=2021. The aging lung: Physiology, disease, and immunity |pdf=|usr=}}{{33811810}} gab.com Text The aging lung: Physiology, disease, and immunity JO: Cell http://www.kidney.de/pget.php?&tw=1&pmid=33811810 #FOAvip The population is aging at a rate never seen before in human history. As the number of elderly adults grows, it is imperative we expand our understanding of the underpinnings of aging biology. Human lungs are composed of a unique panoply of cell types that face ongoing chemical, mechanical, biological, immunological, and xenobiotic stress over a lifetime. Yet, we do not fully appreciate the mechanistic drivers of lung aging and why age increases the risk of parenchymal lung disease, fatal respiratory infection, and primary lung cancer. Here, we review the molecular and cellular aspects of lung aging, local stress response pathways, and how the aging process predisposes to the pathogenesis of pulmonary disease. We place these insights into context of the COVID-19 pandemic and discuss how innate and adaptive immunity within the lung is altered with age. Twit Text FOAVip The aging lung: Physiology, disease, and immunity ????Cell http://www.kidney.de/pget.php?&tw=1&pmid=33811810 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=33811810 #FOAvip English Wikipedia <ref>{{Cite pmid|33811810}}</ref> The aging lung: Physiology, disease, and immunity #MMPMID33811810 Schneider JL; Rowe JH; Garcia-de-Alba C; Kim CF; Sharpe AH; Haigis MC Cell 2021[Apr]; 184 (8): 1990-2019 PMID33811810show ga The population is aging at a rate never seen before in human history. As the number of elderly adults grows, it is imperative we expand our understanding of the underpinnings of aging biology. Human lungs are composed of a unique panoply of cell types that face ongoing chemical, mechanical, biological, immunological, and xenobiotic stress over a lifetime. Yet, we do not fully appreciate the mechanistic drivers of lung aging and why age increases the risk of parenchymal lung disease, fatal respiratory infection, and primary lung cancer. Here, we review the molecular and cellular aspects of lung aging, local stress response pathways, and how the aging process predisposes to the pathogenesis of pulmonary disease. We place these insights into context of the COVID-19 pandemic and discuss how innate and adaptive immunity within the lung is altered with age. |*Aging/immunology/pathology[MESH] |*Cellular Senescence[MESH] |*Lung Diseases/immunology/pathology[MESH] |*Lung/immunology/pathology[MESH] |Adaptive Immunity[MESH] |Aged[MESH] |COVID-19/immunology/pathology[MESH] |Humans[MESH]The aging lung: Physiology, disease, and immunity (epmc).pdf DeepDyve Pubget Overpricing