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10.3390/medsci9010016 http://scihub22266oqcxt.onion/10.3390/medsci9010016 33804308!7931027!33804308     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=33804308&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       Med+Sci+(Basel) 2021 ; 9 (1): ? Nephropedia Template TP {{tp|p=33804308|t=2021. Fibrosis after Myocardial Infarction: An Overview on Cellular Processes, Molecular Pathways, Clinical Evaluation and Prognostic Value |pdf=|usr=}}{{33804308}} gab.com Text Fibrosis after Myocardial Infarction: An Overview on Cellular Processes, Molecular Pathways, Clinical Evaluation and Prognostic Value JO: Med Sci (Basel) http://www.kidney.de/pget.php?&tw=1&pmid=33804308 #FOAvip The ischemic injury caused by myocardial infarction activates a complex healing process wherein a powerful inflammatory response and a reparative phase follow and balance each other. An intricate network of mediators finely orchestrate a large variety of cellular subtypes throughout molecular signaling pathways that determine the intensity and duration of each phase. At the end of this process, the necrotic tissue is replaced with a fibrotic scar whose quality strictly depends on the delicate balance resulting from the interaction between multiple actors involved in fibrogenesis. An inflammatory or reparative dysregulation, both in term of excess and deficiency, may cause ventricular dysfunction and life-threatening arrhythmias that heavily affect clinical outcome. This review discusses cellular process and molecular signaling pathways that determine fibrosis and the imaging technique that can characterize the clinical impact of this process in-vivo. Twit Text FOAVip Fibrosis after Myocardial Infarction: An Overview on Cellular Processes, Molecular Pathways, Clinical Evaluation and Prognostic Value ????Med Sci (Basel) http://www.kidney.de/pget.php?&tw=1&pmid=33804308 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=33804308 #FOAvip English Wikipedia <ref>{{Cite pmid|33804308}}</ref> Fibrosis after Myocardial Infarction: An Overview on Cellular Processes, Molecular Pathways, Clinical Evaluation and Prognostic Value #MMPMID33804308 Scalise RFM; De Sarro R; Caracciolo A; Lauro R; Squadrito F; Carerj S; Bitto A; Micari A; Bella GD; Costa F; Irrera N Med Sci (Basel) 2021[Mar]; 9 (1): ? PMID33804308show ga The ischemic injury caused by myocardial infarction activates a complex healing process wherein a powerful inflammatory response and a reparative phase follow and balance each other. An intricate network of mediators finely orchestrate a large variety of cellular subtypes throughout molecular signaling pathways that determine the intensity and duration of each phase. At the end of this process, the necrotic tissue is replaced with a fibrotic scar whose quality strictly depends on the delicate balance resulting from the interaction between multiple actors involved in fibrogenesis. An inflammatory or reparative dysregulation, both in term of excess and deficiency, may cause ventricular dysfunction and life-threatening arrhythmias that heavily affect clinical outcome. This review discusses cellular process and molecular signaling pathways that determine fibrosis and the imaging technique that can characterize the clinical impact of this process in-vivo. |*Myocardial Infarction/diagnosis[MESH] |Cicatrix[MESH] |Fibrosis[MESH] |Humans[MESH] |Prognosis[MESH]Fibrosis after Myocardial Infarction: An Overview on Cellular Processe(epmc).pdf DeepDyve Pubget Overpricing