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10.3390/ijms22052681 http://scihub22266oqcxt.onion/10.3390/ijms22052681 33800954!7961671!33800954     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=33800954&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Int+J+Mol+Sci 2021 ; 22 (5): ä Nephropedia Template TP {{tp|p=33800954|t=2021. Interactions of SARS-CoV-2 with the Blood-Brain Barrier |pdf=|usr=}}{{33800954}} gab.com Text Interactions of SARS-CoV-2 with the Blood-Brain Barrier JO: Int J Mol Sci http://www.kidney.de/pget.php?&tw=1&pmid=33800954 #FOAvip Emerging data indicate that neurological complications occur as a consequence of severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infection. The blood-brain barrier (BBB) is a critical interface that regulates entry of circulating molecules into the CNS, and is regulated by signals that arise from the brain and blood compartments. In this review, we discuss mechanisms by which SARS-CoV-2 interactions with the BBB may contribute to neurological dysfunction associated with coronavirus disease of 2019 (COVID-19), which is caused by SARS-CoV-2. We consider aspects of peripheral disease, such as hypoxia and systemic inflammatory response syndrome/cytokine storm, as well as CNS infection and mechanisms of viral entry into the brain. We also discuss the contribution of risk factors for developing severe COVID-19 to BBB dysfunction that could increase viral entry or otherwise damage the brain. Twit Text FOAVip Interactions of SARS-CoV-2 with the Blood-Brain Barrier ????Int J Mol Sci http://www.kidney.de/pget.php?&tw=1&pmid=33800954 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=33800954 #FOAvip English Wikipedia <ref>{{Cite pmid|33800954}}</ref> Interactions of SARS-CoV-2 with the Blood-Brain Barrier #MMPMID33800954 Erickson MA; Rhea EM; Knopp RC; Banks WA Int J Mol Sci 2021[Mar]; 22 (5): ä PMID33800954show ga Emerging data indicate that neurological complications occur as a consequence of severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infection. The blood-brain barrier (BBB) is a critical interface that regulates entry of circulating molecules into the CNS, and is regulated by signals that arise from the brain and blood compartments. In this review, we discuss mechanisms by which SARS-CoV-2 interactions with the BBB may contribute to neurological dysfunction associated with coronavirus disease of 2019 (COVID-19), which is caused by SARS-CoV-2. We consider aspects of peripheral disease, such as hypoxia and systemic inflammatory response syndrome/cytokine storm, as well as CNS infection and mechanisms of viral entry into the brain. We also discuss the contribution of risk factors for developing severe COVID-19 to BBB dysfunction that could increase viral entry or otherwise damage the brain. |Animals[MESH] |Blood-Brain Barrier/metabolism/*physiopathology/*virology[MESH] |Brain/virology[MESH] |COVID-19/epidemiology/*virology[MESH] |Central Nervous System Diseases/etiology/virology[MESH] |Comorbidity[MESH] |Humans[MESH] |SARS-CoV-2/chemistry/*metabolism[MESH]Interactions of SARS-CoV-2 with the Blood-Brain Barrier (epmc).pdf DeepDyve Pubget Overpricing