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10.1016/j.tim.2021.03.008

http://scihub22266oqcxt.onion/10.1016/j.tim.2021.03.008
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33795156!8007089!33795156
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suck abstract from ncbi


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pmid33795156      Trends+Microbiol 2021 ; 29 (11): 967-969
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  • Likelihood of amyloid formation in COVID-19-induced ARDS #MMPMID33795156
  • Sinha N; Thakur AK
  • Trends Microbiol 2021[Nov]; 29 (11): 967-969 PMID33795156show ga
  • Severe coronavirus disease 2019 (COVID-19) infection leads to multifactorial acute respiratory distress syndrome (ARDS), with little therapeutic success. The pathophysiology associated with ARDS or post-ARDS is not yet well understood. We hypothesize that amyloid formation occurring due to protein homeostasis disruption can be one of the complications associated with COVID-19-induced-ARDS.
  • |*SARS-CoV-2[MESH]
  • |Amyloid/*metabolism[MESH]
  • |Amyloidosis/etiology/metabolism/pathology[MESH]
  • |Animals[MESH]
  • |COVID-19/*complications/*virology[MESH]
  • |Disease Management[MESH]
  • |Disease Susceptibility[MESH]
  • |Humans[MESH]


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