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10.1016/j.jamcollsurg.2021.02.019 http://scihub22266oqcxt.onion/10.1016/j.jamcollsurg.2021.02.019 33766727!7982779!33766727     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=33766727&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       J+Am+Coll+Surg 2021 ; 232 (6): 995-1003 Nephropedia Template TP {{tp|p=33766727|t=2021. Fibrinolysis Shutdown in COVID-19: Clinical Manifestations, Molecular Mechanisms, and Therapeutic Implications |pdf=|usr=}}{{33766727}} gab.com Text Fibrinolysis Shutdown in COVID-19: Clinical Manifestations, Molecular Mechanisms, and Therapeutic Implications JO: J Am Coll Surg http://www.kidney.de/pget.php?&tw=1&pmid=33766727 #FOAvip The COVID-19 pandemic has introduced a global public health threat unparalleled in our history. The most severe cases are marked by ARDS attributed to microvascular thrombosis. Hypercoagulability, resulting in a profoundly prothrombotic state, is a distinct feature of COVID-19 and is accentuated by a high incidence of fibrinolysis shutdown. The aims of this review were to describe the manifestations of fibrinolysis shutdown in COVID-19 and its associated outcomes, review the molecular mechanisms of dysregulated fibrinolysis associated with COVID-19, and discuss potential implications and therapeutic targets for patients with severe COVID-19. Twit Text FOAVip Fibrinolysis Shutdown in COVID-19: Clinical Manifestations, Molecular Mechanisms, and Therapeutic Implications ????J Am Coll Surg http://www.kidney.de/pget.php?&tw=1&pmid=33766727 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=33766727 #FOAvip English Wikipedia <ref>{{Cite pmid|33766727}}</ref> Fibrinolysis Shutdown in COVID-19: Clinical Manifestations, Molecular Mechanisms, and Therapeutic Implications #MMPMID33766727 Meizoso JP; Moore HB; Moore EE J Am Coll Surg 2021[Jun]; 232 (6): 995-1003 PMID33766727show ga The COVID-19 pandemic has introduced a global public health threat unparalleled in our history. The most severe cases are marked by ARDS attributed to microvascular thrombosis. Hypercoagulability, resulting in a profoundly prothrombotic state, is a distinct feature of COVID-19 and is accentuated by a high incidence of fibrinolysis shutdown. The aims of this review were to describe the manifestations of fibrinolysis shutdown in COVID-19 and its associated outcomes, review the molecular mechanisms of dysregulated fibrinolysis associated with COVID-19, and discuss potential implications and therapeutic targets for patients with severe COVID-19. |*Fibrinolysis[MESH] |Blood Coagulation Disorders/etiology/therapy/virology[MESH] |COVID-19/blood/*complications[MESH] |Humans[MESH]Fibrinolysis Shutdown in COVID-19: Clinical Manifestations, Molecular (epmc).pdf DeepDyve Pubget Overpricing