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10.1007/s12020-021-02671-8

http://scihub22266oqcxt.onion/10.1007/s12020-021-02671-8
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33738708!7972333!33738708
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suck abstract from ncbi


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pmid33738708      Endocrine 2021 ; 72 (1): 1-11
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  • Hypovitaminosis D and the endocrine phenotype of COVID-19 #MMPMID33738708
  • Giustina A
  • Endocrine 2021[Apr]; 72 (1): 1-11 PMID33738708show ga
  • BACKGROUND: Vitamin D and its deficiency have recently been suspected to be involved in increased susceptibility and negative outcomes of COVID-19. This assumption was based on the well known immunomodulatory actions of vitamin D and on the consistent finding of low levels of 25 hydroxyvitamin D (25OHD) in hospitalized patients with COVID-19. Moreover, several studies reported a correlation between 25OHD levels and different clinical outcomes of the disease. AIM: Aim of the current review was to approach the topic of vitamin D and COVID-19 from a different perspective summarizing the data which led to the evidence of the existence of an endocrine phenotype of COVID-19. CONCLUSIONS: This review analyzed in the light of the current knowledge the possibility that several endocrine manifestations of COVID-19 could be holistically interpreted in the context of an inadequate vitamin D status.
  • |*COVID-19[MESH]
  • |*Vitamin D Deficiency/complications[MESH]
  • |Humans[MESH]
  • |Phenotype[MESH]
  • |SARS-CoV-2[MESH]
  • |Vitamin D[MESH]


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