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10.1002/rmv.2234 http://scihub22266oqcxt.onion/10.1002/rmv.2234 33724604!8250219!33724604     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 245.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Rev+Med+Virol 2021 ; 31 (6): e2234 Nephropedia Template TP {{tp|p=33724604|t=2021. Interindividual immunogenic variants: Susceptibility to coronavirus, respiratory syncytial virus and influenza virus |pdf=|usr=}}{{33724604}} gab.com Text Interindividual immunogenic variants: Susceptibility to coronavirus, respiratory syncytial virus and influenza virus JO: Rev Med Virol http://www.kidney.de/pget.php?&tw=1&pmid=33724604 #FOAvip The coronavirus disease (Covid-19) pandemic is the most serious event of the year 2020, causing considerable global morbidity and mortality. The goal of this review is to provide a comprehensive summary of reported associations between inter-individual immunogenic variants and disease susceptibility or symptoms caused by the coronavirus strains severe acute respiratory syndrome-associated coronavirus, severe acute respiratory syndrome-associated coronavirus-2, and two of the main respiratory viruses, respiratory syncytial virus and influenza virus. The results suggest that the genetic background of the host could affect the levels of proinflammatory and anti-inflammatory cytokines and might modulate the progression of Covid-19 in affected patients. Notably, genetic variations in innate immune components such as toll-like receptors and mannose-binding lectin 2 play critical roles in the ability of the immune system to recognize coronavirus and initiate an early immune response to clear the virus and prevent the development of severe symptoms. This review provides promising clues related to the potential benefits of using immunotherapy and immune modulation for respiratory infectious disease treatment in a personalized manner. Twit Text FOAVip Interindividual immunogenic variants: Susceptibility to coronavirus, respiratory syncytial virus and influenza virus ????Rev Med Virol http://www.kidney.de/pget.php?&tw=1&pmid=33724604 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=33724604 #FOAvip English Wikipedia <ref>{{Cite pmid|33724604}}</ref> Interindividual immunogenic variants: Susceptibility to coronavirus, respiratory syncytial virus and influenza virus #MMPMID33724604 Darbeheshti F; Mahdiannasser M; Uhal BD; Ogino S; Gupta S; Rezaei N Rev Med Virol 2021[Nov]; 31 (6): e2234 PMID33724604show ga The coronavirus disease (Covid-19) pandemic is the most serious event of the year 2020, causing considerable global morbidity and mortality. The goal of this review is to provide a comprehensive summary of reported associations between inter-individual immunogenic variants and disease susceptibility or symptoms caused by the coronavirus strains severe acute respiratory syndrome-associated coronavirus, severe acute respiratory syndrome-associated coronavirus-2, and two of the main respiratory viruses, respiratory syncytial virus and influenza virus. The results suggest that the genetic background of the host could affect the levels of proinflammatory and anti-inflammatory cytokines and might modulate the progression of Covid-19 in affected patients. Notably, genetic variations in innate immune components such as toll-like receptors and mannose-binding lectin 2 play critical roles in the ability of the immune system to recognize coronavirus and initiate an early immune response to clear the virus and prevent the development of severe symptoms. This review provides promising clues related to the potential benefits of using immunotherapy and immune modulation for respiratory infectious disease treatment in a personalized manner. |*Genetic Predisposition to Disease[MESH] |Antiviral Agents/therapeutic use[MESH] |Biological Variation, Individual[MESH] |COVID-19 Drug Treatment[MESH] |COVID-19/genetics/*immunology/virology[MESH] |Cytokine Release Syndrome/drug therapy/genetics/*immunology/virology[MESH] |Gene Expression[MESH] |Humans[MESH] |Immunity, Innate[MESH] |Immunologic Factors/therapeutic use[MESH] |Influenza, Human/drug therapy/genetics/*immunology/virology[MESH] |Mannose-Binding Lectin/genetics/immunology[MESH] |Orthomyxoviridae/drug effects/immunology[MESH] |Respiratory Syncytial Virus Infections/drug therapy/genetics/*immunology/virology[MESH] |Respiratory Syncytial Viruses/drug effects/immunology[MESH] |SARS-CoV-2/classification/drug effects/immunology[MESH] |Severe Acute Respiratory Syndrome/drug therapy/genetics/*immunology/virology[MESH] |Severe acute respiratory syndrome-related coronavirus/drug effects/immunology[MESH]Interindividual immunogenic variants: Susceptibility to coronavirus, r(epmc).pdf DeepDyve Pubget Overpricing