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10.3389/fphys.2020.588664 http://scihub22266oqcxt.onion/10.3389/fphys.2020.588664 33716756!7947867!33716756     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=33716756&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 500 Internal Server Error in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       Front+Physiol 2020 ; 11 (ä): 588664 Nephropedia Template TP {{tp|p=33716756|t=2020. Sodium Transporters in Human Health and Disease |pdf=|usr=}}{{33716756}} gab.com Text Sodium Transporters in Human Health and Disease JO: Front Physiol http://www.kidney.de/pget.php?&tw=1&pmid=33716756 #FOAvip Sodium (Na(+)) electrochemical gradients established by Na(+)/K(+) ATPase activity drives the transport of ions, minerals, and sugars in both excitable and non-excitable cells. Na(+)-dependent transporters can move these solutes in the same direction (cotransport) or in opposite directions (exchanger) across both the apical and basolateral plasma membranes of polarized epithelia. In addition to maintaining physiological homeostasis of these solutes, increases and decreases in sodium may also initiate, directly or indirectly, signaling cascades that regulate a variety of intracellular post-translational events. In this review, we will describe how the Na(+)/K(+) ATPase maintains a Na(+) gradient utilized by multiple sodium-dependent transport mechanisms to regulate glucose uptake, excitatory neurotransmitters, calcium signaling, acid-base balance, salt-wasting disorders, fluid volume, and magnesium transport. We will discuss how several Na(+)-dependent cotransporters and Na(+)-dependent exchangers have significant roles in human health and disease. Finally, we will discuss how each of these Na(+)-dependent transport mechanisms have either been shown or have the potential to use Na(+) in a secondary role as a signaling molecule. Twit Text FOAVip Sodium Transporters in Human Health and Disease ????Front Physiol http://www.kidney.de/pget.php?&tw=1&pmid=33716756 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=33716756 #FOAvip English Wikipedia <ref>{{Cite pmid|33716756}}</ref> Sodium Transporters in Human Health and Disease #MMPMID33716756 Gagnon KB; Delpire E Front Physiol 2020[]; 11 (ä): 588664 PMID33716756show ga Sodium (Na(+)) electrochemical gradients established by Na(+)/K(+) ATPase activity drives the transport of ions, minerals, and sugars in both excitable and non-excitable cells. Na(+)-dependent transporters can move these solutes in the same direction (cotransport) or in opposite directions (exchanger) across both the apical and basolateral plasma membranes of polarized epithelia. In addition to maintaining physiological homeostasis of these solutes, increases and decreases in sodium may also initiate, directly or indirectly, signaling cascades that regulate a variety of intracellular post-translational events. In this review, we will describe how the Na(+)/K(+) ATPase maintains a Na(+) gradient utilized by multiple sodium-dependent transport mechanisms to regulate glucose uptake, excitatory neurotransmitters, calcium signaling, acid-base balance, salt-wasting disorders, fluid volume, and magnesium transport. We will discuss how several Na(+)-dependent cotransporters and Na(+)-dependent exchangers have significant roles in human health and disease. Finally, we will discuss how each of these Na(+)-dependent transport mechanisms have either been shown or have the potential to use Na(+) in a secondary role as a signaling molecule. äSodium Transporters in Human Health and Disease (epmc).pdf DeepDyve Pubget Overpricing