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10.1002/eji.202048984 http://scihub22266oqcxt.onion/10.1002/eji.202048984 33687066!8250324!33687066     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Eur+J+Immunol 2021 ; 51 (5): 1062-1070 Nephropedia Template TP {{tp|p=33687066|t=2021. Insights into coronavirus immunity taught by the murine coronavirus |pdf=|usr=}}{{33687066}} gab.com Text Insights into coronavirus immunity taught by the murine coronavirus JO: Eur J Immunol http://www.kidney.de/pget.php?&tw=1&pmid=33687066 #FOAvip Coronaviruses (CoVs) represent enveloped, ss RNA viruses with the ability to infect a range of vertebrates causing mainly lung, CNS, enteric, and hepatic disease. While the infection with human CoV is commonly associated with mild respiratory symptoms, the emergence of SARS-CoV, MERS-CoV, and SARS-CoV-2 highlights the potential for CoVs to cause severe respiratory and systemic disease. The devastating global health burden caused by SARS-CoV-2 has spawned countless studies seeking clinical correlates of disease severity and host susceptibility factors, revealing a complex network of antiviral immune circuits. The mouse hepatitis virus (MHV) is, like SARS-CoV-2, a beta-CoV and is endemic in wild mice. Laboratory MHV strains have been extensively studied to reveal coronavirus virulence factors and elucidate host mechanisms of antiviral immunity. These are reviewed here with the aim to identify translational insights for SARS-CoV-2 learned from murine CoVs. Twit Text FOAVip Insights into coronavirus immunity taught by the murine coronavirus ????Eur J Immunol http://www.kidney.de/pget.php?&tw=1&pmid=33687066 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=33687066 #FOAvip English Wikipedia <ref>{{Cite pmid|33687066}}</ref> Insights into coronavirus immunity taught by the murine coronavirus #MMPMID33687066 Grabherr S; Ludewig B; Pikor NB Eur J Immunol 2021[May]; 51 (5): 1062-1070 PMID33687066show ga Coronaviruses (CoVs) represent enveloped, ss RNA viruses with the ability to infect a range of vertebrates causing mainly lung, CNS, enteric, and hepatic disease. While the infection with human CoV is commonly associated with mild respiratory symptoms, the emergence of SARS-CoV, MERS-CoV, and SARS-CoV-2 highlights the potential for CoVs to cause severe respiratory and systemic disease. The devastating global health burden caused by SARS-CoV-2 has spawned countless studies seeking clinical correlates of disease severity and host susceptibility factors, revealing a complex network of antiviral immune circuits. The mouse hepatitis virus (MHV) is, like SARS-CoV-2, a beta-CoV and is endemic in wild mice. Laboratory MHV strains have been extensively studied to reveal coronavirus virulence factors and elucidate host mechanisms of antiviral immunity. These are reviewed here with the aim to identify translational insights for SARS-CoV-2 learned from murine CoVs. |Adaptive Immunity/*immunology[MESH] |Animals[MESH] |Coronavirus Infections/*immunology/*pathology[MESH] |Disease Models, Animal[MESH] |Humans[MESH] |Mice[MESH] |Middle East Respiratory Syndrome Coronavirus/immunology[MESH] |Murine hepatitis virus/*immunology/*pathogenicity[MESH] |SARS-CoV-2/immunology[MESH] |Severe acute respiratory syndrome-related coronavirus/immunology[MESH] |Severity of Illness Index[MESH] |Spike Glycoprotein, Coronavirus/metabolism[MESH]Insights into coronavirus immunity taught by the murine coronavirus (epmc).pdf DeepDyve Pubget Overpricing