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10.1016/j.jacbts.2021.01.002 http://scihub22266oqcxt.onion/10.1016/j.jacbts.2021.01.002 33681537!7909907!33681537     free     free     free Deprecated: Implicit conversion from float 227.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 227.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 227.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 227.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       JACC+Basic+Transl+Sci 2021 ; 6 (4): 331-345 Nephropedia Template TP {{tp|p=33681537|t=2021. SARS-CoV-2 Infects Human Engineered Heart Tissues and Models COVID-19 Myocarditis |pdf=|usr=}}{{33681537}} gab.com Text SARS-CoV-2 Infects Human Engineered Heart Tissues and Models COVID-19 Myocarditis JO: JACC Basic Transl Sci http://www.kidney.de/pget.php?&tw=1&pmid=33681537 #FOAvip There is ongoing debate as to whether cardiac complications of coronavirus disease-2019 (COVID-19) result from myocardial viral infection or are secondary to systemic inflammation and/or thrombosis. We provide evidence that cardiomyocytes are infected in patients with COVID-19 myocarditis and are susceptible to severe acute respiratory syndrome coronavirus 2. We establish an engineered heart tissue model of COVID-19 myocardial pathology, define mechanisms of viral pathogenesis, and demonstrate that cardiomyocyte severe acute respiratory syndrome coronavirus 2 infection results in contractile deficits, cytokine production, sarcomere disassembly, and cell death. These findings implicate direct infection of cardiomyocytes in the pathogenesis of COVID-19 myocardial pathology and provides a model system to study this emerging disease. Twit Text FOAVip SARS-CoV-2 Infects Human Engineered Heart Tissues and Models COVID-19 Myocarditis ????JACC Basic Transl Sci http://www.kidney.de/pget.php?&tw=1&pmid=33681537 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=33681537 #FOAvip English Wikipedia <ref>{{Cite pmid|33681537}}</ref> SARS-CoV-2 Infects Human Engineered Heart Tissues and Models COVID-19 Myocarditis #MMPMID33681537 Bailey AL; Dmytrenko O; Greenberg L; Bredemeyer AL; Ma P; Liu J; Penna V; Winkler ES; Sviben S; Brooks E; Nair AP; Heck KA; Rali AS; Simpson L; Saririan M; Hobohm D; Stump WT; Fitzpatrick JA; Xie X; Zhang X; Shi PY; Hinson JT; Gi WT; Schmidt C; Leuschner F; Lin CY; Diamond MS; Greenberg MJ; Lavine KJ JACC Basic Transl Sci 2021[Apr]; 6 (4): 331-345 PMID33681537show ga There is ongoing debate as to whether cardiac complications of coronavirus disease-2019 (COVID-19) result from myocardial viral infection or are secondary to systemic inflammation and/or thrombosis. We provide evidence that cardiomyocytes are infected in patients with COVID-19 myocarditis and are susceptible to severe acute respiratory syndrome coronavirus 2. We establish an engineered heart tissue model of COVID-19 myocardial pathology, define mechanisms of viral pathogenesis, and demonstrate that cardiomyocyte severe acute respiratory syndrome coronavirus 2 infection results in contractile deficits, cytokine production, sarcomere disassembly, and cell death. These findings implicate direct infection of cardiomyocytes in the pathogenesis of COVID-19 myocardial pathology and provides a model system to study this emerging disease. äSARS-CoV-2 Infects Human Engineered Heart Tissues and Models COVID-19 (epmc).pdf DeepDyve Pubget Overpricing