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10.1530/VB-20-0017 http://scihub22266oqcxt.onion/10.1530/VB-20-0017 33659858!7923034!33659858     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=33659858&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       Vasc+Biol 2021 ; 3 (1): R15-R23 Nephropedia Template TP {{tp|p=33659858|t=2021. SARS-CoV-2 and endothelial cell interaction in COVID-19: molecular perspectives |pdf=|usr=}}{{33659858}} gab.com Text SARS-CoV-2 and endothelial cell interaction in COVID-19: molecular perspectives JO: Vasc Biol http://www.kidney.de/pget.php?&tw=1&pmid=33659858 #FOAvip SARS-CoV-2 is the agent responsible for the coronavirus disease (COVID-19), which has been declared a pandemic by the World Health Organization. The clinical evolution of COVID-19 ranges from asymptomatic infection to death. Older people and patients with underlying medical conditions, particularly diabetes, cardiovascular and chronic respiratory diseases are more susceptible to develop severe forms of COVID-19. Significant endothelial damage has been reported in COVID-19 and growing evidence supports the key pathophysiological role of this alteration in the onset and the progression of the disease. In particular, the impaired vascular homeostasis secondary to the structural and functional damage of the endothelium and its main component, the endothelial cells, contributes to the systemic proinflammatory state and the multiorgan involvement observed in COVID-19 patients. This review summarizes the current evidence supporting the proposition that the endothelium is a key target of SARS-CoV-2, with a focus on the molecular mechanisms involved in the interaction between SARS-CoV-2 and endothelial cells. Twit Text FOAVip SARS-CoV-2 and endothelial cell interaction in COVID-19: molecular perspectives ????Vasc Biol http://www.kidney.de/pget.php?&tw=1&pmid=33659858 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=33659858 #FOAvip English Wikipedia <ref>{{Cite pmid|33659858}}</ref> SARS-CoV-2 and endothelial cell interaction in COVID-19: molecular perspectives #MMPMID33659858 Giordo R; Paliogiannis P; Mangoni AA; Pintus G Vasc Biol 2021[]; 3 (1): R15-R23 PMID33659858show ga SARS-CoV-2 is the agent responsible for the coronavirus disease (COVID-19), which has been declared a pandemic by the World Health Organization. The clinical evolution of COVID-19 ranges from asymptomatic infection to death. Older people and patients with underlying medical conditions, particularly diabetes, cardiovascular and chronic respiratory diseases are more susceptible to develop severe forms of COVID-19. Significant endothelial damage has been reported in COVID-19 and growing evidence supports the key pathophysiological role of this alteration in the onset and the progression of the disease. In particular, the impaired vascular homeostasis secondary to the structural and functional damage of the endothelium and its main component, the endothelial cells, contributes to the systemic proinflammatory state and the multiorgan involvement observed in COVID-19 patients. This review summarizes the current evidence supporting the proposition that the endothelium is a key target of SARS-CoV-2, with a focus on the molecular mechanisms involved in the interaction between SARS-CoV-2 and endothelial cells. ?SARS-CoV-2 and endothelial cell interaction in COVID-19: molecular per(epmc).pdf DeepDyve Pubget Overpricing