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10.1016/j.drudis.2021.02.009 http://scihub22266oqcxt.onion/10.1016/j.drudis.2021.02.009 33609782!7889466!33609782     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=33609782&cmd=llinks): Failed to open stream: HTTP request failed! 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Inflammation control and improvement of cognitive function in COVID-19 infections: is there a role for kynurenine 3-monooxygenase inhibition?|pdf=|usr=}}{{33609782}} gab.com Text Inflammation control and improvement of cognitive function in COVID-19 infections: is there a role for kynurenine 3-monooxygenase inhibition JO: Drug Discov Today http://www.kidney.de/pget.php?&tw=1&pmid=33609782 #FOAvip The novel respiratory virus severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), which causes coronavirus disease 2019 (COVID-19), emerged during late 2019 and spread rapidly across the world. It is now recognised that the nervous system can be affected in COVID-19, with several studies reporting long-term cognitive problems in patients. The metabolic pathway of tryptophan degradation, known as the kynurenine pathway (KP), is significantly activated in patients with COVID-19. KP metabolites have roles in regulating both inflammatory/immune responses and neurological functions. In this review, we speculate on the effects of KP activation in patients with COVID-19, and how modulation of this pathway might impact inflammation and reduce neurological symptoms. Twit Text FOAVip Inflammation control and improvement of cognitive function in COVID-19 infections: is there a role for kynurenine 3-monooxygenase inhibition ????Drug Discov Today http://www.kidney.de/pget.php?&tw=1&pmid=33609782 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=33609782 #FOAvip English Wikipedia <ref>{{Cite pmid|33609782}}</ref> Inflammation control and improvement of cognitive function in COVID-19 infections: is there a role for kynurenine 3-monooxygenase inhibition? #MMPMID33609782 Collier ME; Zhang S; Scrutton NS; Giorgini F Drug Discov Today 2021[Jun]; 26 (6): 1473-1481 PMID33609782show ga The novel respiratory virus severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), which causes coronavirus disease 2019 (COVID-19), emerged during late 2019 and spread rapidly across the world. It is now recognised that the nervous system can be affected in COVID-19, with several studies reporting long-term cognitive problems in patients. The metabolic pathway of tryptophan degradation, known as the kynurenine pathway (KP), is significantly activated in patients with COVID-19. KP metabolites have roles in regulating both inflammatory/immune responses and neurological functions. In this review, we speculate on the effects of KP activation in patients with COVID-19, and how modulation of this pathway might impact inflammation and reduce neurological symptoms. |*COVID-19/immunology/psychology[MESH] |*Cognition/drug effects/physiology[MESH] |Animals[MESH] |Humans[MESH] |Inflammation/*metabolism[MESH] |Kynurenine 3-Monooxygenase/antagonists & inhibitors[MESH] |Kynurenine/*metabolism[MESH] |Neuroprotective Agents/pharmacology[MESH] |Signal Transduction[MESH] |Sulfonamides/*pharmacology[MESH] |Thiazoles/*pharmacology[MESH]Inflammation control and improvement of cognitive function in COVID-19(epmc).pdf DeepDyve Pubget Overpricing