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10.1016/j.drudis.2021.02.009

http://scihub22266oqcxt.onion/10.1016/j.drudis.2021.02.009
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33609782!7889466!33609782
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suck abstract from ncbi


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pmid33609782      Drug+Discov+Today 2021 ; 26 (6): 1473-1481
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  • Inflammation control and improvement of cognitive function in COVID-19 infections: is there a role for kynurenine 3-monooxygenase inhibition? #MMPMID33609782
  • Collier ME; Zhang S; Scrutton NS; Giorgini F
  • Drug Discov Today 2021[Jun]; 26 (6): 1473-1481 PMID33609782show ga
  • The novel respiratory virus severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), which causes coronavirus disease 2019 (COVID-19), emerged during late 2019 and spread rapidly across the world. It is now recognised that the nervous system can be affected in COVID-19, with several studies reporting long-term cognitive problems in patients. The metabolic pathway of tryptophan degradation, known as the kynurenine pathway (KP), is significantly activated in patients with COVID-19. KP metabolites have roles in regulating both inflammatory/immune responses and neurological functions. In this review, we speculate on the effects of KP activation in patients with COVID-19, and how modulation of this pathway might impact inflammation and reduce neurological symptoms.
  • |*COVID-19/immunology/psychology[MESH]
  • |*Cognition/drug effects/physiology[MESH]
  • |Animals[MESH]
  • |Humans[MESH]
  • |Inflammation/*metabolism[MESH]
  • |Kynurenine 3-Monooxygenase/antagonists & inhibitors[MESH]
  • |Kynurenine/*metabolism[MESH]
  • |Neuroprotective Agents/pharmacology[MESH]
  • |Signal Transduction[MESH]
  • |Sulfonamides/*pharmacology[MESH]
  • |Thiazoles/*pharmacology[MESH]


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