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10.1016/j.cytogfr.2021.01.003 http://scihub22266oqcxt.onion/10.1016/j.cytogfr.2021.01.003 33608189!7871890!33608189     free     free     free Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 247.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 247.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Cytokine+Growth+Factor+Rev 2021 ; 58 (ä): 55-65 Nephropedia Template TP {{tp|p=33608189|t=2021. Type I and III IFN-mediated antiviral actions counteracted by SARS-CoV-2 proteins and host inherited factors |pdf=|usr=}}{{33608189}} gab.com Text Type I and III IFN-mediated antiviral actions counteracted by SARS-CoV-2 proteins and host inherited factors JO: Cytokine Growth Factor Rev http://www.kidney.de/pget.php?&tw=1&pmid=33608189 #FOAvip SARS-CoV-2 is a recently identified coronavirus accountable for the current pandemic disease known as COVID-19. Different patterns of disease progression infer a diverse host immune response, with interferon (IFN) being pivotal. IFN-I and III are produced and released by virus-infected cells during the interplay with SARS-CoV-2, thus establishing an antiviral state in target cells. However, the efficacy of IFN and its role in the possible outcomes of the disease are not yet defined, as it is influenced both by factors inherent to the virus and to the host. The virus exhibits multiple strategies to counteract the innate immune response, including those shared by SARS-CoV and MERS-CoV and other novel ones. Inborn errors in the host may affect IFN-related effector proteins or decrease its levels in plasma upon neutralization by preexistent autoantibodies. This battle between the IFN response triggered upon SARS-CoV-2 infection, its magnitude and timing, and the efficacy of its antiviral tools in dispute against the viral evasion strategies together with the genetic factors of the host, generate a scenario whose fate contributes to defining the severity of COVID-19. Twit Text FOAVip Type I and III IFN-mediated antiviral actions counteracted by SARS-CoV-2 proteins and host inherited factors ????Cytokine Growth Factor Rev http://www.kidney.de/pget.php?&tw=1&pmid=33608189 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=33608189 #FOAvip English Wikipedia <ref>{{Cite pmid|33608189}}</ref> Type I and III IFN-mediated antiviral actions counteracted by SARS-CoV-2 proteins and host inherited factors #MMPMID33608189 Quarleri J; Delpino MV Cytokine Growth Factor Rev 2021[Apr]; 58 (ä): 55-65 PMID33608189show ga SARS-CoV-2 is a recently identified coronavirus accountable for the current pandemic disease known as COVID-19. Different patterns of disease progression infer a diverse host immune response, with interferon (IFN) being pivotal. IFN-I and III are produced and released by virus-infected cells during the interplay with SARS-CoV-2, thus establishing an antiviral state in target cells. However, the efficacy of IFN and its role in the possible outcomes of the disease are not yet defined, as it is influenced both by factors inherent to the virus and to the host. The virus exhibits multiple strategies to counteract the innate immune response, including those shared by SARS-CoV and MERS-CoV and other novel ones. Inborn errors in the host may affect IFN-related effector proteins or decrease its levels in plasma upon neutralization by preexistent autoantibodies. This battle between the IFN response triggered upon SARS-CoV-2 infection, its magnitude and timing, and the efficacy of its antiviral tools in dispute against the viral evasion strategies together with the genetic factors of the host, generate a scenario whose fate contributes to defining the severity of COVID-19. |*Host-Pathogen Interactions/genetics/immunology[MESH] |Animals[MESH] |Antiviral Agents/metabolism[MESH] |COVID-19/genetics/immunology/pathology[MESH] |Genetic Diseases, Inborn/complications/immunology[MESH] |Genetic Predisposition to Disease[MESH] |Humans[MESH] |Immune Evasion/genetics/immunology[MESH] |Immunity, Innate/genetics[MESH] |Interferon Lambda[MESH] |Interferon Type I/antagonists & inhibitors/*physiology[MESH] |Interferons/antagonists & inhibitors/*physiology[MESH] |Pandemics[MESH] |SARS-CoV-2/*immunology/pathogenicity[MESH]Type I and III IFN-mediated antiviral actions counteracted by SARS-CoV(epmc).pdf DeepDyve Pubget Overpricing