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10.1016/j.pnpbp.2021.110276 http://scihub22266oqcxt.onion/10.1016/j.pnpbp.2021.110276 33549696!ä!33549696 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Prog+Neuropsychopharmacol+Biol+Psychiatry 2021 ; 109 (ä): 110276 Nephropedia Template TP {{tp|p=33549696|t=2021. A role for pathogen risk factors and autoimmunity in encephalitis lethargica?|pdf=|usr=}}{{33549696}} gab.com Text A role for pathogen risk factors and autoimmunity in encephalitis lethargica JO: Prog Neuropsychopharmacol Biol Psychiatry http://www.kidney.de/pget.php?&tw=1&pmid=33549696 #FOAvip The encephalitis lethargica (EL) epidemic swept the world from 1916 to 1926 and is estimated to have afflicted between 80,000 to one million people. EL is an unusual neurological illness that causes profound sleep disorders, devastating neurological sequalae and, in many cases, death. Though uncommon, EL is still occasionally diagnosed today when a patient presents with an acute or subacute encephalitic illness, where all other known causes of encephalitis have been excluded and criteria for EL are met. However, it is impossible to know whether recent cases of EL-like syndromes result from the same disease that caused the epidemic. After more than 100 years of research into potential pathogen triggers and the role of autoimmune processes, the aetiology of EL remains unknown. The epidemic approximately coincided with the 1918 H1N1 influenza pandemic but the evidence of a causal link is inconclusive. This article reviews the literature on the causes of EL with a focus on autoimmune mechanisms. In light of the current pandemic, we also consider the parallels between the EL epidemic and neurological manifestations of COVID-19. Understanding how pathogens and autoimmune processes can affect the brain may well help us understand the conundrum of EL and, more importantly, will guide the treatment of patients with suspected COVID-19-related neurological disease, as well as prepare us for any future epidemic of a neurological illness. Twit Text FOAVip A role for pathogen risk factors and autoimmunity in encephalitis lethargica ????Prog Neuropsychopharmacol Biol Psychiatry http://www.kidney.de/pget.php?&tw=1&pmid=33549696 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=33549696 #FOAvip English Wikipedia <ref>{{Cite pmid|33549696}}</ref> A role for pathogen risk factors and autoimmunity in encephalitis lethargica? #MMPMID33549696 Bond M; Bechter K; Muller N; Tebartz van Elst L; Meier UC Prog Neuropsychopharmacol Biol Psychiatry 2021[Jul]; 109 (ä): 110276 PMID33549696show ga The encephalitis lethargica (EL) epidemic swept the world from 1916 to 1926 and is estimated to have afflicted between 80,000 to one million people. EL is an unusual neurological illness that causes profound sleep disorders, devastating neurological sequalae and, in many cases, death. Though uncommon, EL is still occasionally diagnosed today when a patient presents with an acute or subacute encephalitic illness, where all other known causes of encephalitis have been excluded and criteria for EL are met. However, it is impossible to know whether recent cases of EL-like syndromes result from the same disease that caused the epidemic. After more than 100 years of research into potential pathogen triggers and the role of autoimmune processes, the aetiology of EL remains unknown. The epidemic approximately coincided with the 1918 H1N1 influenza pandemic but the evidence of a causal link is inconclusive. This article reviews the literature on the causes of EL with a focus on autoimmune mechanisms. In light of the current pandemic, we also consider the parallels between the EL epidemic and neurological manifestations of COVID-19. Understanding how pathogens and autoimmune processes can affect the brain may well help us understand the conundrum of EL and, more importantly, will guide the treatment of patients with suspected COVID-19-related neurological disease, as well as prepare us for any future epidemic of a neurological illness. |Autoimmune Diseases/history/*immunology/*virology[MESH] |COVID-19[MESH] |Encephalitis/history/*immunology/*virology[MESH] |History, 20th Century[MESH] |Humans[MESH] |Pandemics/*history[MESH]A role for pathogen risk factors and autoimmunity in encephalitis leth(epmc).pdf DeepDyve Pubget Overpricing