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10.1159/000512815 http://scihub22266oqcxt.onion/10.1159/000512815 33530091!7900470!33530091     free     free     free Deprecated: Implicit conversion from float 229.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 229.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 229.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 229.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 229.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Neuroimmunomodulation 2021 ; 28 (1): 22-28 Nephropedia Template TP {{tp|p=33530091|t=2021. Neuroinflammation and Brain Development: Possible Risk Factors in COVID-19-Infected Children |pdf=|usr=}}{{33530091}} gab.com Text Neuroinflammation and Brain Development: Possible Risk Factors in COVID-19-Infected Children JO: Neuroimmunomodulation http://www.kidney.de/pget.php?&tw=1&pmid=33530091 #FOAvip COVID-19, a disease caused by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) betacoronavirus, affects children in a different way than it does in adults, with milder symptoms. However, several cases of neurological symptoms with neuroinflammatory syndromes, such as the multisystem inflammatory syndrome (MIS-C), following mild cases, have been reported. As with other viral infections, such as rubella, influenza, and cytomegalovirus, SARS-CoV-2 induces a surge of proinflammatory cytokines that affect microglial function, which can be harmful to brain development. Along with the viral induction of neuroinflammation, other noninfectious conditions may interact to produce additional inflammation, such as the nutritional imbalance of fatty acids and polyunsaturated fatty acids and alcohol consumption during pregnancy. Additionally, transient thyrotoxicosis induced by SARS-CoV-2 with secondary autoimmune hypothyroidism has been reported, which could go undetected during pregnancy. Together, those factors may pose additional risk factors for SARS-CoV-2 infection impacting mechanisms of neural development such as synaptic pruning and neural circuitry formation. The present review discusses those conditions in the perspective of the understanding of risk factors that should be considered and the possible emergence of neurodevelopmental disorders in COVID-19-infected children. Twit Text FOAVip Neuroinflammation and Brain Development: Possible Risk Factors in COVID-19-Infected Children ????Neuroimmunomodulation http://www.kidney.de/pget.php?&tw=1&pmid=33530091 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=33530091 #FOAvip English Wikipedia <ref>{{Cite pmid|33530091}}</ref> Neuroinflammation and Brain Development: Possible Risk Factors in COVID-19-Infected Children #MMPMID33530091 da Silva Chagas L; Sandre PC; de Velasco PC; Marcondes H; Ribeiro E Ribeiro NCA; Barreto AL; Alves Mauro LB; Ferreira JH; Serfaty CA Neuroimmunomodulation 2021[]; 28 (1): 22-28 PMID33530091show ga COVID-19, a disease caused by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) betacoronavirus, affects children in a different way than it does in adults, with milder symptoms. However, several cases of neurological symptoms with neuroinflammatory syndromes, such as the multisystem inflammatory syndrome (MIS-C), following mild cases, have been reported. As with other viral infections, such as rubella, influenza, and cytomegalovirus, SARS-CoV-2 induces a surge of proinflammatory cytokines that affect microglial function, which can be harmful to brain development. Along with the viral induction of neuroinflammation, other noninfectious conditions may interact to produce additional inflammation, such as the nutritional imbalance of fatty acids and polyunsaturated fatty acids and alcohol consumption during pregnancy. Additionally, transient thyrotoxicosis induced by SARS-CoV-2 with secondary autoimmune hypothyroidism has been reported, which could go undetected during pregnancy. Together, those factors may pose additional risk factors for SARS-CoV-2 infection impacting mechanisms of neural development such as synaptic pruning and neural circuitry formation. The present review discusses those conditions in the perspective of the understanding of risk factors that should be considered and the possible emergence of neurodevelopmental disorders in COVID-19-infected children. |Brain/*growth & development/immunology/physiopathology[MESH] |COVID-19/*immunology/physiopathology[MESH] |Diet[MESH] |Dietary Fats, Unsaturated[MESH] |Fatty Acids, Unsaturated[MESH] |Fetal Alcohol Spectrum Disorders/immunology/physiopathology[MESH] |Humans[MESH] |Inflammation/*immunology/physiopathology[MESH] |Microglia/*immunology[MESH] |Neurodevelopmental Disorders/*immunology/physiopathology[MESH] |Neuronal Plasticity[MESH] |Risk Factors[MESH] |SARS-CoV-2[MESH] |Severity of Illness Index[MESH]Neuroinflammation and Brain Development: Possible Risk Factors in COV(epmc).pdf DeepDyve Pubget Overpricing