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10.23750/abm.v91i4.10768 http://scihub22266oqcxt.onion/10.23750/abm.v91i4.10768 33525240!7927483!33525240     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Acta+Biomed 2020 ; 91 (4): e2020138 Nephropedia Template TP {{tp|p=33525240|t=2020. Hepcidin is a friend rather than a foe in COVID19-induced complications |pdf=|usr=}}{{33525240}} gab.com Text Hepcidin is a friend rather than a foe in COVID19-induced complications JO: Acta Biomed http://www.kidney.de/pget.php?&tw=1&pmid=33525240 #FOAvip Clinical observations in concert with literary data demonstrate that detrimental complications of COVID19-induced pathology (acute respiratory distress syndrome, multi-organ failure, Kawasaki-like disease etc.), could result from a disturbance of local iron homeostasis (FeH) in damaged tissues followed by abnormal coagulation in small vessels. To resolve these complications the local FeH needs to be recovered. Hepcidin, as a master regulator of FeH is both a major player in the recovery and a marker of an efficacy of the restoration. Therefore, both local and systemic hepcidin levels could serve as a dynamic marker of disease progression (the more hepcidin the worse is disease) and treatment efficacy (after iron homeostasis is recovered hepcidin disappears). On the contrast, artificial attempts to suppress hepcidin expression directly or application of hepcidin antagonists could be detrimental. Overall, more comprehensive research of hepcidin role in COVID-19 pathology is needed. Twit Text FOAVip Hepcidin is a friend rather than a foe in COVID19-induced complications ????Acta Biomed http://www.kidney.de/pget.php?&tw=1&pmid=33525240 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=33525240 #FOAvip English Wikipedia <ref>{{Cite pmid|33525240}}</ref> Hepcidin is a friend rather than a foe in COVID19-induced complications #MMPMID33525240 Sukhomlin T Acta Biomed 2020[Nov]; 91 (4): e2020138 PMID33525240show ga Clinical observations in concert with literary data demonstrate that detrimental complications of COVID19-induced pathology (acute respiratory distress syndrome, multi-organ failure, Kawasaki-like disease etc.), could result from a disturbance of local iron homeostasis (FeH) in damaged tissues followed by abnormal coagulation in small vessels. To resolve these complications the local FeH needs to be recovered. Hepcidin, as a master regulator of FeH is both a major player in the recovery and a marker of an efficacy of the restoration. Therefore, both local and systemic hepcidin levels could serve as a dynamic marker of disease progression (the more hepcidin the worse is disease) and treatment efficacy (after iron homeostasis is recovered hepcidin disappears). On the contrast, artificial attempts to suppress hepcidin expression directly or application of hepcidin antagonists could be detrimental. Overall, more comprehensive research of hepcidin role in COVID-19 pathology is needed. |*COVID-19[MESH] |*Diabetes Mellitus[MESH] |*Iron Overload[MESH] |Aged[MESH] |Friends[MESH] |Hepcidins[MESH] |Humans[MESH] |Obesity[MESH]Hepcidin is a friend rather than a foe in COVID19-induced complication(epmc).pdf DeepDyve Pubget Overpricing