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10.1002/jmv.26826

http://scihub22266oqcxt.onion/10.1002/jmv.26826
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33506952!8014260!33506952
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suck abstract from ncbi


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pmid33506952      J+Med+Virol 2021 ; 93 (5): 2735-2739
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  • Role of Toll-like receptors in the pathogenesis of COVID-19 #MMPMID33506952
  • Khanmohammadi S; Rezaei N
  • J Med Virol 2021[May]; 93 (5): 2735-2739 PMID33506952show ga
  • Coronavirus disease 2019 (COVID-19), caused by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), has led to a pandemic since March 2020. The exact pathogenesis of SARS-CoV-2 and the role of each component of the innate and adaptive immune system is still unknown. However, available data from other coronavirus families, such as SARS-CoV and the Middle East respiratory syndrome and also new findings could be useful for a better understanding of SARS-CoV-2. Toll-like receptors (TLR) play an important role in recognition of viral particles and activation of the innate immune system. Activation of TLR pathways leads to secretion of pro-inflammatory cytokines, such as interleukin-1 (IL-1), IL-6, and tumor necrosis factor-alpha, as well as type 1 interferon. Different TLRs, like TLR2, TLR3, TLR4, TLR6, TLR7, TLR8, and TLR9 are potentially important in COVID-19 infection. It is also worth mentioning that we should bear in mind both the beneficial and harmful effects of TLR in confronting COVID-19 infection. TLRs could be a potential target in controlling the infection in the early stages of disease and production of vaccine against SARS-CoV-2.
  • |Adaptive Immunity[MESH]
  • |Animals[MESH]
  • |COVID-19 Drug Treatment[MESH]
  • |COVID-19/immunology/metabolism/*pathology[MESH]
  • |Cytokines/metabolism[MESH]
  • |Humans[MESH]
  • |Immunity, Innate[MESH]
  • |Inflammation[MESH]
  • |SARS-CoV-2/metabolism/*pathogenicity[MESH]
  • |Signal Transduction[MESH]


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