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10.1177/1179551420984125

http://scihub22266oqcxt.onion/10.1177/1179551420984125
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33488135!7768872!33488135
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suck abstract from ncbi


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pmid33488135      Clin+Med+Insights+Endocrinol+Diabetes 2020 ; 13 (ä): 1179551420984125
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  • Diabetic Ketoacidosis on Hospitalization with COVID-19 in a Previously Nondiabetic Patient: A Review of Pathophysiology #MMPMID33488135
  • Eskandarani RM; Sawan S
  • Clin Med Insights Endocrinol Diabetes 2020[]; 13 (ä): 1179551420984125 PMID33488135show ga
  • Hyperglycaemia during inpatient admission is indicative of higher morbidity and mortality risks in critically ill patients. The severe acute respiratory distress coronavirus 2 (SARS-CoV-2) has been reported to induce ketoacidosis and diabetic ketoacidosis (DKA) even in nondiabetic patients. The pathophysiology of the SARS-CoV-2 infection that can contribute to hyperglycaemia, and the exacerbated inflammatory cytokine storm can overlap with the metabolic chronic inflammatory state attributable to the metabolic syndrome, which underlies diabetes mellitus. In this report, we explore the possible pathophysiology and metabolic mechanisms that lead to metabolic acidosis in nondiabetic patients.
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