Use my Search Websuite to scan PubMed, PMCentral, Journal Hosts and Journal Archives, FullText. Kick-your-searchterm to multiple Engines kick-your-query now !>

A dictionary by aggregated review articles of nephrology, medicine and the life sciences Your one-stop-run pathway from word to the immediate pdf of peer-reviewed on-topic knowledge.

10.1007/s11481-020-09980-1 http://scihub22266oqcxt.onion/10.1007/s11481-020-09980-1 33462776!7813171!33462776     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       J+Neuroimmune+Pharmacol 2021 ; 16 (1): 48-58 Nephropedia Template TP {{tp|p=33462776|t=2021. Purinergic Signaling of ATP in COVID-19 Associated Guillain-Barre Syndrome |pdf=|usr=}}{{33462776}} gab.com Text Purinergic Signaling of ATP in COVID-19 Associated Guillain-Barre Syndrome JO: J Neuroimmune Pharmacol http://www.kidney.de/pget.php?&tw=1&pmid=33462776 #FOAvip Declared as a global public health emergency, coronavirus disease 2019 (COVID-19) is presented as a disease of the respiratory tract, although severe cases can affect the entire organism. Several studies have shown neurological symptoms, ranging from dizziness and loss of consciousness to cerebrovascular and neurodegenerative diseases. In this context, Guillain-Barre syndrome, an immune-mediated inflammatory neuropathy, has been closely associated with critical cases of infection with "severe acute respiratory syndrome of coronavirus 2" (SARS-CoV-2), the etiological agent of COVID-19. Its pathophysiology is related to a generalized inflammation that affects the nervous system, but neurotropism was also revealed by the new coronavirus, which may increase the risk of neurological sequel, as well as the mortality of the disease. Thus, considering the comorbidities that SARS-CoV-2 infection can promote, the modulation of purinergic signaling can be applied as a potential therapy. In this perspective, given the role of adenosine triphosphate (ATP) in neural intercommunication, the P2X7 receptor (P2X7R) acts on microglia cells and its inhibition may be able to reduce the inflammatory condition of neurodegenerative diseases. Finally, alternative measures to circumvent the reality of the COVID-19 pandemic need to be considered, given the severity of critical cases and the viral involvement of multiple organs. Twit Text FOAVip Purinergic Signaling of ATP in COVID-19 Associated Guillain-Barre Syndrome ????J Neuroimmune Pharmacol http://www.kidney.de/pget.php?&tw=1&pmid=33462776 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=33462776 #FOAvip English Wikipedia <ref>{{Cite pmid|33462776}}</ref> Purinergic Signaling of ATP in COVID-19 Associated Guillain-Barre Syndrome #MMPMID33462776 Simoes JLB; Bagatini MD J Neuroimmune Pharmacol 2021[Mar]; 16 (1): 48-58 PMID33462776show ga Declared as a global public health emergency, coronavirus disease 2019 (COVID-19) is presented as a disease of the respiratory tract, although severe cases can affect the entire organism. Several studies have shown neurological symptoms, ranging from dizziness and loss of consciousness to cerebrovascular and neurodegenerative diseases. In this context, Guillain-Barre syndrome, an immune-mediated inflammatory neuropathy, has been closely associated with critical cases of infection with "severe acute respiratory syndrome of coronavirus 2" (SARS-CoV-2), the etiological agent of COVID-19. Its pathophysiology is related to a generalized inflammation that affects the nervous system, but neurotropism was also revealed by the new coronavirus, which may increase the risk of neurological sequel, as well as the mortality of the disease. Thus, considering the comorbidities that SARS-CoV-2 infection can promote, the modulation of purinergic signaling can be applied as a potential therapy. In this perspective, given the role of adenosine triphosphate (ATP) in neural intercommunication, the P2X7 receptor (P2X7R) acts on microglia cells and its inhibition may be able to reduce the inflammatory condition of neurodegenerative diseases. Finally, alternative measures to circumvent the reality of the COVID-19 pandemic need to be considered, given the severity of critical cases and the viral involvement of multiple organs. |*Adenosine Triphosphate[MESH] |*Receptors, Purinergic[MESH] |*Signal Transduction[MESH] |COVID-19/*complications[MESH] |Guillain-Barre Syndrome/*etiology/*physiopathology[MESH] |Humans[MESH]Purinergic Signaling of ATP in COVID-19 Associated Guillain-Barre Synd(epmc).pdf DeepDyve Pubget Overpricing