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10.1016/j.biopha.2021.111233 http://scihub22266oqcxt.onion/10.1016/j.biopha.2021.111233 33433350!7834878!33433350     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Biomed+Pharmacother 2021 ; 135 (ä): 111233 Nephropedia Template TP {{tp|p=33433350|t=2021. COVID-19 and inflammatory bowel disease: A pathophysiological assessment |pdf=|usr=}}{{33433350}} gab.com Text COVID-19 and inflammatory bowel disease: A pathophysiological assessment JO: Biomed Pharmacother http://www.kidney.de/pget.php?&tw=1&pmid=33433350 #FOAvip Coronavirus disease-2019 (COVID-19), caused by SARS-CoV-2, has led to the ongoing global pandemic. Although most patients experience no or only mild symptoms, some patients can develop severe illness, such as progressive pneumonia, acute respiratory distress syndrome, secondary hemophagocytic lymphohistiocytosis and multiple organ failure caused by cytokine release syndrome. A majority of COVID-19 patients also develop gastrointestinal symptoms. These can present special challenges to the management of patients with inflammatory bowel disease (IBD) due to potential interactions between the immune response related to SARS-CoV-2 infection and dysregulated immunity associated with IBD. In this context, the pathogenesis of COVID-19 is reviewed in order to address these questions regarding immune interactions between COVID-19 and IBD. Twit Text FOAVip COVID-19 and inflammatory bowel disease: A pathophysiological assessment ????Biomed Pharmacother http://www.kidney.de/pget.php?&tw=1&pmid=33433350 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=33433350 #FOAvip English Wikipedia <ref>{{Cite pmid|33433350}}</ref> COVID-19 and inflammatory bowel disease: A pathophysiological assessment #MMPMID33433350 Yang C; Xiao SY Biomed Pharmacother 2021[Mar]; 135 (ä): 111233 PMID33433350show ga Coronavirus disease-2019 (COVID-19), caused by SARS-CoV-2, has led to the ongoing global pandemic. Although most patients experience no or only mild symptoms, some patients can develop severe illness, such as progressive pneumonia, acute respiratory distress syndrome, secondary hemophagocytic lymphohistiocytosis and multiple organ failure caused by cytokine release syndrome. A majority of COVID-19 patients also develop gastrointestinal symptoms. These can present special challenges to the management of patients with inflammatory bowel disease (IBD) due to potential interactions between the immune response related to SARS-CoV-2 infection and dysregulated immunity associated with IBD. In this context, the pathogenesis of COVID-19 is reviewed in order to address these questions regarding immune interactions between COVID-19 and IBD. |Angiotensin-Converting Enzyme 2/drug effects/immunology[MESH] |Anti-Inflammatory Agents/pharmacology/therapeutic use[MESH] |COVID-19 Drug Treatment[MESH] |COVID-19/*epidemiology/immunology/*physiopathology[MESH] |Cytokine Release Syndrome/drug therapy/epidemiology/immunology/physiopathology[MESH] |Humans[MESH] |Immunity/drug effects/*physiology[MESH] |Immunosuppressive Agents/pharmacology/therapeutic use[MESH] |Inflammatory Bowel Diseases/drug therapy/*epidemiology/immunology/*physiopathology[MESH]COVID-19 and inflammatory bowel disease: A pathophysiological assessme(epmc).pdf DeepDyve Pubget Overpricing