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10.3390/jcm10020191 http://scihub22266oqcxt.onion/10.3390/jcm10020191 33430431!7827226!33430431     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       J+Clin+Med 2021 ; 10 (2): ä Nephropedia Template TP {{tp|p=33430431|t=2021. Proposal of the Definition for COVID-19-Associated Coagulopathy |pdf=|usr=}}{{33430431}} gab.com Text Proposal of the Definition for COVID-19-Associated Coagulopathy JO: J Clin Med http://www.kidney.de/pget.php?&tw=1&pmid=33430431 #FOAvip Thrombotic events are common complications in COVID-19 patients that include both thrombus formation in large vessels and the microvasculature of the lung and other organs. COVID-19-associated coagulopathy (CAC) and disseminated intravascular coagulation (DIC) have similarities and differences, and whether CAC is a form of DIC is the subject of debate. Reported mechanisms of CAC include activated coagulation, endotheliopathy, up-regulated innate and adaptive immunity, and activated complement system. Although the clinical features and laboratory findings of CAC and DIC seem different, there are fundamental similarities that should be considered. Basically, the pathological findings of COVID-19 fall within the scope of the definition of DIC, i.e., systemic activation of coagulation caused by or resulting from the microvascular damage. Therefore, we suggest that although CAC differs from usual infection-associated DIC, its various features indicate that it can be considered a thrombotic phenotype DIC. This review summarizes the current knowledge about CAC including differences and similarities with sepsis-associated DIC. Twit Text FOAVip Proposal of the Definition for COVID-19-Associated Coagulopathy ????J Clin Med http://www.kidney.de/pget.php?&tw=1&pmid=33430431 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=33430431 #FOAvip English Wikipedia <ref>{{Cite pmid|33430431}}</ref> Proposal of the Definition for COVID-19-Associated Coagulopathy #MMPMID33430431 Iba T; Warkentin TE; Thachil J; Levi M; Levy JH J Clin Med 2021[Jan]; 10 (2): ä PMID33430431show ga Thrombotic events are common complications in COVID-19 patients that include both thrombus formation in large vessels and the microvasculature of the lung and other organs. COVID-19-associated coagulopathy (CAC) and disseminated intravascular coagulation (DIC) have similarities and differences, and whether CAC is a form of DIC is the subject of debate. Reported mechanisms of CAC include activated coagulation, endotheliopathy, up-regulated innate and adaptive immunity, and activated complement system. Although the clinical features and laboratory findings of CAC and DIC seem different, there are fundamental similarities that should be considered. Basically, the pathological findings of COVID-19 fall within the scope of the definition of DIC, i.e., systemic activation of coagulation caused by or resulting from the microvascular damage. Therefore, we suggest that although CAC differs from usual infection-associated DIC, its various features indicate that it can be considered a thrombotic phenotype DIC. This review summarizes the current knowledge about CAC including differences and similarities with sepsis-associated DIC. äProposal of the Definition for COVID-19-Associated Coagulopathy (epmc).pdf DeepDyve Pubget Overpricing