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10.3390/ijms22010323 http://scihub22266oqcxt.onion/10.3390/ijms22010323 33396899!7796338!33396899     free     free     free Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Int+J+Mol+Sci 2020 ; 22 (1): ä Nephropedia Template TP {{tp|p=33396899|t=2020. Role of Host-Mediated Post-Translational Modifications (PTMs) in RNA Virus Pathogenesis |pdf=|usr=}}{{33396899}} gab.com Text Role of Host-Mediated Post-Translational Modifications (PTMs) in RNA Virus Pathogenesis JO: Int J Mol Sci http://www.kidney.de/pget.php?&tw=1&pmid=33396899 #FOAvip Being opportunistic intracellular pathogens, viruses are dependent on the host for their replication. They hijack host cellular machinery for their replication and survival by targeting crucial cellular physiological pathways, including transcription, translation, immune pathways, and apoptosis. Immediately after translation, the host and viral proteins undergo a process called post-translational modification (PTM). PTMs of proteins involves the attachment of small proteins, carbohydrates/lipids, or chemical groups to the proteins and are crucial for the proteins' functioning. During viral infection, host proteins utilize PTMs to control the virus replication, using strategies like activating immune response pathways, inhibiting viral protein synthesis, and ultimately eliminating the virus from the host. PTM of viral proteins increases solubility, enhances antigenicity and virulence properties. However, RNA viruses are devoid of enzymes capable of introducing PTMs to their proteins. Hence, they utilize the host PTM machinery to promote their survival. Proteins from viruses belonging to the family: Togaviridae, Flaviviridae, Retroviridae, and Coronaviridae such as chikungunya, dengue, zika, HIV, and coronavirus are a few that are well-known to be modified. This review discusses various host and virus-mediated PTMs that play a role in the outcome during the infection. Twit Text FOAVip Role of Host-Mediated Post-Translational Modifications (PTMs) in RNA Virus Pathogenesis ????Int J Mol Sci http://www.kidney.de/pget.php?&tw=1&pmid=33396899 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=33396899 #FOAvip English Wikipedia <ref>{{Cite pmid|33396899}}</ref> Role of Host-Mediated Post-Translational Modifications (PTMs) in RNA Virus Pathogenesis #MMPMID33396899 Kumar R; Mehta D; Mishra N; Nayak D; Sunil S Int J Mol Sci 2020[Dec]; 22 (1): ä PMID33396899show ga Being opportunistic intracellular pathogens, viruses are dependent on the host for their replication. They hijack host cellular machinery for their replication and survival by targeting crucial cellular physiological pathways, including transcription, translation, immune pathways, and apoptosis. Immediately after translation, the host and viral proteins undergo a process called post-translational modification (PTM). PTMs of proteins involves the attachment of small proteins, carbohydrates/lipids, or chemical groups to the proteins and are crucial for the proteins' functioning. During viral infection, host proteins utilize PTMs to control the virus replication, using strategies like activating immune response pathways, inhibiting viral protein synthesis, and ultimately eliminating the virus from the host. PTM of viral proteins increases solubility, enhances antigenicity and virulence properties. However, RNA viruses are devoid of enzymes capable of introducing PTMs to their proteins. Hence, they utilize the host PTM machinery to promote their survival. Proteins from viruses belonging to the family: Togaviridae, Flaviviridae, Retroviridae, and Coronaviridae such as chikungunya, dengue, zika, HIV, and coronavirus are a few that are well-known to be modified. This review discusses various host and virus-mediated PTMs that play a role in the outcome during the infection. |*Protein Processing, Post-Translational[MESH] |Acetylation[MESH] |Chikungunya virus/metabolism[MESH] |Coronavirus/metabolism/pathogenicity[MESH] |Cytopathogenic Effect, Viral[MESH] |Glycosylation[MESH] |HIV/metabolism/pathogenicity[MESH] |Host Microbial Interactions[MESH] |Humans[MESH] |Phosphorylation[MESH] |RNA Virus Infections/*enzymology/immunology/metabolism/*virology[MESH] |RNA Viruses/immunology/*metabolism/*pathogenicity[MESH] |Ubiquitination[MESH] |Viral Proteins/*metabolism[MESH] |Virus Replication/physiology[MESH]Role of Host-Mediated Post-Translational Modifications (PTMs) in RNA V(epmc).pdf DeepDyve Pubget Overpricing