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10.1089/jir.2020.0214 http://scihub22266oqcxt.onion/10.1089/jir.2020.0214 33337934!7757701!33337934     free     free     free Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       J+Interferon+Cytokine+Res 2020 ; 40 (12): 543-548 Nephropedia Template TP {{tp|p=33337934|t=2020. Antagonism of Type I Interferon by Severe Acute Respiratory Syndrome Coronavirus 2 |pdf=|usr=}}{{33337934}} gab.com Text Antagonism of Type I Interferon by Severe Acute Respiratory Syndrome Coronavirus 2 JO: J Interferon Cytokine Res http://www.kidney.de/pget.php?&tw=1&pmid=33337934 #FOAvip The coronavirus disease 2019 (COVID-19) pandemic is caused by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), warranting urgent study of the molecular mechanisms of SARS-CoV-2 infection and host immune response. Type I interferon (IFN-I) is a key component of host innate immune system responsible for eliminating the virus at the early stage of infection. In contrast, SARS-CoV-2 has evolved multiple strategies to evade innate immune response to facilitate viral replication, transmission, and pathogenesis. This review summarizes the recent progresses on SARS-CoV-2 proteins that antagonize host IFN-I production and/or signaling. These progresses have provided knowledge for new vaccine and antiviral development to prevent and control COVID-19. Twit Text FOAVip Antagonism of Type I Interferon by Severe Acute Respiratory Syndrome Coronavirus 2 ????J Interferon Cytokine Res http://www.kidney.de/pget.php?&tw=1&pmid=33337934 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=33337934 #FOAvip English Wikipedia <ref>{{Cite pmid|33337934}}</ref> Antagonism of Type I Interferon by Severe Acute Respiratory Syndrome Coronavirus 2 #MMPMID33337934 Xia H; Shi PY J Interferon Cytokine Res 2020[Dec]; 40 (12): 543-548 PMID33337934show ga The coronavirus disease 2019 (COVID-19) pandemic is caused by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), warranting urgent study of the molecular mechanisms of SARS-CoV-2 infection and host immune response. Type I interferon (IFN-I) is a key component of host innate immune system responsible for eliminating the virus at the early stage of infection. In contrast, SARS-CoV-2 has evolved multiple strategies to evade innate immune response to facilitate viral replication, transmission, and pathogenesis. This review summarizes the recent progresses on SARS-CoV-2 proteins that antagonize host IFN-I production and/or signaling. These progresses have provided knowledge for new vaccine and antiviral development to prevent and control COVID-19. |*Immunity, Innate[MESH] |Antiviral Agents/therapeutic use[MESH] |COVID-19 Vaccines[MESH] |COVID-19/*immunology[MESH] |Humans[MESH] |Immune System[MESH] |Interferon Type I/*immunology[MESH] |Mutation[MESH] |Phosphorylation[MESH] |SARS-CoV-2/*immunology/physiology[MESH] |Signal Transduction[MESH] |Viral Nonstructural Proteins/*immunology[MESH]Antagonism of Type I Interferon by Severe Acute Respiratory Syndrome C(epmc).pdf DeepDyve Pubget Overpricing