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10.1016/j.mehy.2020.110419 http://scihub22266oqcxt.onion/10.1016/j.mehy.2020.110419 33309251!7683957!33309251     free     free     free Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Med+Hypotheses 2021 ; 146 (ä): 110419 Nephropedia Template TP {{tp|p=33309251|t=2021. Is the oral cavity a reservoir for prolonged SARS-CoV-2 shedding?|pdf=|usr=}}{{33309251}} gab.com Text Is the oral cavity a reservoir for prolonged SARS-CoV-2 shedding JO: Med Hypotheses http://www.kidney.de/pget.php?&tw=1&pmid=33309251 #FOAvip Limited knowledge about the contagiosity and case fatality rate of COVID-19 as well as the still enigmatic route of transmission have led to strict limitations of non-emergency health care especially in head and neck medicine and dentistry. There are theories that the oral cavity provides a favorable environment for SARS-CoV-2 entry and persistence which may be a risk for prolonged virus shedding. However, intraoral innate immune mechanisms provide antiviral effects against a myriad of pathogenic viruses. Initial hints of their efficacy against SARS-CoV-2 are surfacing. It is hypothesized that intraoral immune system activity modulates the invasion pattern of SARS-CoV-2 into oral cells. Thus, the significance of intraoral tissues for SARS-CoV-2 transmission and persistence cannot be assessed. The underlying concept for this hypothesis was developed by the critical observation of a clinically asymptomatic COVID-19 patient. Despite a positive throat swab for SARS-CoV-2, molecular pathologic analysis of an oral perisulcular tissue specimen failed to detect SARS-CoV-2 RNA. More research effort is necessary to define the true origin of the contagiosity of asymptomatic COVID-19 patients. Twit Text FOAVip Is the oral cavity a reservoir for prolonged SARS-CoV-2 shedding ????Med Hypotheses http://www.kidney.de/pget.php?&tw=1&pmid=33309251 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=33309251 #FOAvip English Wikipedia <ref>{{Cite pmid|33309251}}</ref> Is the oral cavity a reservoir for prolonged SARS-CoV-2 shedding? #MMPMID33309251 Troeltzsch M; Berndt R; Troeltzsch M Med Hypotheses 2021[Jan]; 146 (ä): 110419 PMID33309251show ga Limited knowledge about the contagiosity and case fatality rate of COVID-19 as well as the still enigmatic route of transmission have led to strict limitations of non-emergency health care especially in head and neck medicine and dentistry. There are theories that the oral cavity provides a favorable environment for SARS-CoV-2 entry and persistence which may be a risk for prolonged virus shedding. However, intraoral innate immune mechanisms provide antiviral effects against a myriad of pathogenic viruses. Initial hints of their efficacy against SARS-CoV-2 are surfacing. It is hypothesized that intraoral immune system activity modulates the invasion pattern of SARS-CoV-2 into oral cells. Thus, the significance of intraoral tissues for SARS-CoV-2 transmission and persistence cannot be assessed. The underlying concept for this hypothesis was developed by the critical observation of a clinically asymptomatic COVID-19 patient. Despite a positive throat swab for SARS-CoV-2, molecular pathologic analysis of an oral perisulcular tissue specimen failed to detect SARS-CoV-2 RNA. More research effort is necessary to define the true origin of the contagiosity of asymptomatic COVID-19 patients. |*Models, Biological[MESH] |*Virus Shedding[MESH] |Adult[MESH] |Asymptomatic Infections[MESH] |COVID-19 Nucleic Acid Testing[MESH] |COVID-19/diagnosis/immunology/*virology[MESH] |Disease Reservoirs/virology[MESH] |Female[MESH] |Host Microbial Interactions/immunology[MESH] |Humans[MESH] |Immunity, Innate[MESH] |Mouth/immunology/*virology[MESH] |Pandemics[MESH] |RNA, Viral/isolation & purification[MESH] |SARS-CoV-2/*pathogenicity/*physiology[MESH]Is the oral cavity a reservoir for prolonged SARS-CoV-2 shedding?(epmc).pdf DeepDyve Pubget Overpricing