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10.1186/s42234-020-00058-0

http://scihub22266oqcxt.onion/10.1186/s42234-020-00058-0
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33292846!7683278!33292846
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suck abstract from ncbi


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pmid33292846      Bioelectron+Med 2020 ; 6 (1): 22
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  • Autonomic balance determines the severity of COVID-19 courses #MMPMID33292846
  • Leitzke M; Stefanovic D; Meyer JJ; Schimpf S; Schonknecht P
  • Bioelectron Med 2020[Nov]; 6 (1): 22 PMID33292846show ga
  • COVID-19 has left mankind desperately seeking how to manage dramatically rising infection rates associated with severe disease progressions. COVID-19 courses range from mild symptoms up to multiple organ failure and death, triggered by excessively high serum cytokine levels (IL 1beta, IL 6, TNF alpha, IL 8). The vagally driven cholinergic anti-inflammatory pathway (CAP) stops the action of nuclear factor kappaB (NF-kappaB), the transcriptional factor of pro-inflammatory cytokines. Thus, well-balanced cytokine release depends on adequate vagal signaling. Coronaviruses replicate using NF-kappaB transcriptional factor as well. By degrading the cytoplasmatic inhibitor of NF-kappaB subunits (IkappaB), coronaviruses induce unrestricted NF-kappaB expression accelerating both, virus replication and cytokine transcription.We hypothesize that CAP detriment due to depressed vagal tone critically determines the severity of COVID-19.
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