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10.5005/jp-journals-10071-23547 http://scihub22266oqcxt.onion/10.5005/jp-journals-10071-23547 33281323!7689135!33281323     free     free     free Deprecated: Implicit conversion from float 229.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 229.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Indian+J+Crit+Care+Med 2020 ; 24 (10): 967-970 Nephropedia Template TP {{tp|p=33281323|t=2020. Mechanisms of Hypoxia in COVID-19 Patients: A Pathophysiologic Reflection |pdf=|usr=}}{{33281323}} gab.com Text Mechanisms of Hypoxia in COVID-19 Patients: A Pathophysiologic Reflection JO: Indian J Crit Care Med http://www.kidney.de/pget.php?&tw=1&pmid=33281323 #FOAvip Coronavirus disease-2019 (COVID-19) causes severe hypoxemia which fulfills the criteria of acute respiratory distress syndrome (ARDS) but is not accompanied by typical features of the syndrome. The combination of factors including low P/F ratios, high A-a gradient, relatively preserved lung mechanics, and normal pulmonary pressures may imply a process occurring on the vascular side of the alveolar-capillary unit. The scant but rapidly evolving data available on the pathophysiology are seemingly conflicting, indicating the relative dominance of intrapulmonary shunting or dead space in different studies. In this hypothesis paper, we attempt to gather and explain these observations within a unified conceptual framework by invoking the relative contributions of microvascular thrombosis, along with two proposed vascular mechanisms of capillary flow redistribution and flow through intrapulmonary arteriovenous anastomoses (IPAVA). How to cite this article: Nitsure M, Sarangi B, Shankar GH, Reddy VS, Walimbe A, Sharma V, et al. Mechanisms of Hypoxia in COVID-19 Patients: A Pathophysiologic Reflection. Indian J Crit Care Med 2020;24(10):967-970. Twit Text FOAVip Mechanisms of Hypoxia in COVID-19 Patients: A Pathophysiologic Reflection ????Indian J Crit Care Med http://www.kidney.de/pget.php?&tw=1&pmid=33281323 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=33281323 #FOAvip English Wikipedia <ref>{{Cite pmid|33281323}}</ref> Mechanisms of Hypoxia in COVID-19 Patients: A Pathophysiologic Reflection #MMPMID33281323 Nitsure M; Sarangi B; Shankar GH; Reddy VS; Walimbe A; Sharma V; Prayag S Indian J Crit Care Med 2020[Oct]; 24 (10): 967-970 PMID33281323show ga Coronavirus disease-2019 (COVID-19) causes severe hypoxemia which fulfills the criteria of acute respiratory distress syndrome (ARDS) but is not accompanied by typical features of the syndrome. The combination of factors including low P/F ratios, high A-a gradient, relatively preserved lung mechanics, and normal pulmonary pressures may imply a process occurring on the vascular side of the alveolar-capillary unit. The scant but rapidly evolving data available on the pathophysiology are seemingly conflicting, indicating the relative dominance of intrapulmonary shunting or dead space in different studies. In this hypothesis paper, we attempt to gather and explain these observations within a unified conceptual framework by invoking the relative contributions of microvascular thrombosis, along with two proposed vascular mechanisms of capillary flow redistribution and flow through intrapulmonary arteriovenous anastomoses (IPAVA). How to cite this article: Nitsure M, Sarangi B, Shankar GH, Reddy VS, Walimbe A, Sharma V, et al. Mechanisms of Hypoxia in COVID-19 Patients: A Pathophysiologic Reflection. Indian J Crit Care Med 2020;24(10):967-970. äMechanisms of Hypoxia in COVID-19 Patients: A Pathophysiologic Reflect(epmc).pdf DeepDyve Pubget Overpricing