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10.3389/fcell.2020.586487 http://scihub22266oqcxt.onion/10.3389/fcell.2020.586487 33224951!7674171!33224951     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=33224951&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       Front+Cell+Dev+Biol 2020 ; 8 (?): 586487 Nephropedia Template TP {{tp|p=33224951|t=2020. Roles of TRAFs in Ischemia-Reperfusion Injury |pdf=|usr=}}{{33224951}} gab.com Text Roles of TRAFs in Ischemia-Reperfusion Injury JO: Front Cell Dev Biol http://www.kidney.de/pget.php?&tw=1&pmid=33224951 #FOAvip Tumor necrosis factor receptor-associated factor (TRAF) proteins are a family of signaling molecules that function downstream of multiple receptor signaling pathways, and they play a pivotal role in the regulation of intracellular biological progresses. These TRAF-dependent signaling pathways and physiological functions have been involved in the occurrence and progression of ischemia-reperfusion injury (IRI), which is a common pathophysiological process that occurs in a wide variety of clinical events, including ischemic shock, organ transplantation, and thrombolytic therapy, resulting in a poor prognosis and high mortality. IRI occurs in multiple organs, including liver, kidney, heart, lung, brain, intestine, and retina. In recent years, mounting compelling evidence has confirmed that the genetic alterations of TRAFs can cause subversive phenotype changes during IRI of those organs. In this review, based on current knowledge, we summarized and analyzed the regulatory effect of TRAFs on the IRI of various organs, providing clear direction and a firm theoretical basis for the development of treatment strategies to manipulate TRAF proteins or TRAF-dependent signaling pathways in IRI-related diseases. Twit Text FOAVip Roles of TRAFs in Ischemia-Reperfusion Injury ????Front Cell Dev Biol http://www.kidney.de/pget.php?&tw=1&pmid=33224951 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=33224951 #FOAvip English Wikipedia <ref>{{Cite pmid|33224951}}</ref> Roles of TRAFs in Ischemia-Reperfusion Injury #MMPMID33224951 Zhou W; Lin D; Zhong Z; Ye Q Front Cell Dev Biol 2020[]; 8 (?): 586487 PMID33224951show ga Tumor necrosis factor receptor-associated factor (TRAF) proteins are a family of signaling molecules that function downstream of multiple receptor signaling pathways, and they play a pivotal role in the regulation of intracellular biological progresses. These TRAF-dependent signaling pathways and physiological functions have been involved in the occurrence and progression of ischemia-reperfusion injury (IRI), which is a common pathophysiological process that occurs in a wide variety of clinical events, including ischemic shock, organ transplantation, and thrombolytic therapy, resulting in a poor prognosis and high mortality. IRI occurs in multiple organs, including liver, kidney, heart, lung, brain, intestine, and retina. In recent years, mounting compelling evidence has confirmed that the genetic alterations of TRAFs can cause subversive phenotype changes during IRI of those organs. In this review, based on current knowledge, we summarized and analyzed the regulatory effect of TRAFs on the IRI of various organs, providing clear direction and a firm theoretical basis for the development of treatment strategies to manipulate TRAF proteins or TRAF-dependent signaling pathways in IRI-related diseases. ?Roles of TRAFs in Ischemia-Reperfusion Injury (epmc).pdf DeepDyve Pubget Overpricing