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10.1111/dth.14565

http://scihub22266oqcxt.onion/10.1111/dth.14565
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33219572!7744832!33219572
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suck abstract from ncbi


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pmid33219572      Dermatol+Ther 2021 ; 34 (1): e14565
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  • From dermatological conditions to COVID-19: Reasoning for anticoagulation, suppression of inflammation, and hyperbaric oxygen therapy #MMPMID33219572
  • Criado PR; Miot HA; Pincelli TPH; Fabro AT
  • Dermatol Ther 2021[Jan]; 34 (1): e14565 PMID33219572show ga
  • COVID-19 generates a complex systemic inflammatory response that can lead to death due to wide macrophage activation, endothelial damage, and coagulation in critically ill patients. SARS-CoV-2-induced lung injury due to inflammatory mediated thrombosis could be similar to the livedoid vasculopathy in the skin, supporting a translational comparison of these clinical settings. In this article, we discuss anticoagulation, suppression of inflammatory response, and hyperbaric oxygen therapy in the context of severe COVID-19 and livedoid vasculopathy.
  • |*COVID-19/complications[MESH]
  • |*Hyperbaric Oxygenation[MESH]
  • |Anticoagulants/adverse effects[MESH]
  • |Heparin, Low-Molecular-Weight[MESH]
  • |Humans[MESH]
  • |Inflammation/therapy[MESH]
  • |SARS-CoV-2[MESH]


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