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10.1038/s41467-020-19706-9

http://scihub22266oqcxt.onion/10.1038/s41467-020-19706-9
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33203833!7673112!33203833
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suck abstract from ncbi


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pmid33203833      Nat+Commun 2020 ; 11 (1): 5859
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  • Immune suppression in the early stage of COVID-19 disease #MMPMID33203833
  • Tian W; Zhang N; Jin R; Feng Y; Wang S; Gao S; Gao R; Wu G; Tian D; Tan W; Chen Y; Gao GF; Wong CCL
  • Nat Commun 2020[Nov]; 11 (1): 5859 PMID33203833show ga
  • The outbreak of COVID-19 has become a worldwide pandemic. The pathogenesis of this infectious disease and how it differs from other drivers of pneumonia is unclear. Here we analyze urine samples from COVID-19 infection cases, healthy donors and non-COVID-19 pneumonia cases using quantitative proteomics. The molecular changes suggest that immunosuppression and tight junction impairment occur in the early stage of COVID-19 infection. Further subgrouping of COVID-19 patients into moderate and severe types shows that an activated immune response emerges in severely affected patients. We propose a two-stage mechanism of pathogenesis for this unusual viral infection. Our data advance our understanding of the clinical features of COVID-19 infections and provide a resource for future mechanistic and therapeutics studies.
  • |Betacoronavirus/pathogenicity[MESH]
  • |Biomarkers/urine[MESH]
  • |COVID-19[MESH]
  • |Coronavirus Infections/*immunology/*pathology/urine[MESH]
  • |Disease Progression[MESH]
  • |Humans[MESH]
  • |Immune Tolerance[MESH]
  • |Pandemics[MESH]
  • |Pneumonia, Viral/*immunology/*pathology/urine[MESH]
  • |Pneumonia/immunology/pathology/urine[MESH]
  • |Proteome/analysis[MESH]
  • |SARS-CoV-2[MESH]


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