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10.1134/S0006297920100065 http://scihub22266oqcxt.onion/10.1134/S0006297920100065 33202203!7590568!33202203     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=33202203&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       Biochemistry+(Mosc) 2020 ; 85 (10): 1178-1190 Nephropedia Template TP {{tp|p=33202203|t=2020. NETosis: Molecular Mechanisms, Role in Physiology and Pathology |pdf=|usr=}}{{33202203}} gab.com Text NETosis: Molecular Mechanisms, Role in Physiology and Pathology JO: Biochemistry (Mosc) http://www.kidney.de/pget.php?&tw=1&pmid=33202203 #FOAvip NETosis is a program for formation of neutrophil extracellular traps (NETs), which consist of modified chromatin decorated with bactericidal proteins from granules and cytoplasm. Various pathogens, antibodies and immune complexes, cytokines, microcrystals, and other physiological stimuli can cause NETosis. Induction of NETosis depends on reactive oxygen species (ROS), the main source of which is NADPH oxidase. Activation of NADPH oxidase depends on increase in the concentration of Ca2+ in the cytoplasm and in some cases on the generation of ROS in mitochondria. NETosis includes release of the granule components into the cytosol, modification of histones leading to chromatin decondensation, destruction of the nuclear envelope, as well as formation of pores in the plasma membrane. In this review, basic mechanisms of NETosis, as well as its role in the pathogenesis of some diseases including COVID-19 are discussed. Twit Text FOAVip NETosis: Molecular Mechanisms, Role in Physiology and Pathology ????Biochemistry (Mosc) http://www.kidney.de/pget.php?&tw=1&pmid=33202203 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=33202203 #FOAvip English Wikipedia <ref>{{Cite pmid|33202203}}</ref> NETosis: Molecular Mechanisms, Role in Physiology and Pathology #MMPMID33202203 Vorobjeva NV; Chernyak BV Biochemistry (Mosc) 2020[Oct]; 85 (10): 1178-1190 PMID33202203show ga NETosis is a program for formation of neutrophil extracellular traps (NETs), which consist of modified chromatin decorated with bactericidal proteins from granules and cytoplasm. Various pathogens, antibodies and immune complexes, cytokines, microcrystals, and other physiological stimuli can cause NETosis. Induction of NETosis depends on reactive oxygen species (ROS), the main source of which is NADPH oxidase. Activation of NADPH oxidase depends on increase in the concentration of Ca2+ in the cytoplasm and in some cases on the generation of ROS in mitochondria. NETosis includes release of the granule components into the cytosol, modification of histones leading to chromatin decondensation, destruction of the nuclear envelope, as well as formation of pores in the plasma membrane. In this review, basic mechanisms of NETosis, as well as its role in the pathogenesis of some diseases including COVID-19 are discussed. |*SARS-CoV-2[MESH] |COVID-19/*immunology/*pathology/virology[MESH] |Calcium/metabolism[MESH] |Chromatin/metabolism[MESH] |Extracellular Traps/*immunology/*metabolism[MESH] |Histones/metabolism[MESH] |Humans[MESH] |Mitochondria/metabolism[MESH] |NADPH Oxidases/metabolism[MESH] |Neutrophils/immunology[MESH] |Oxidative Stress/immunology[MESH]NETosis: Molecular Mechanisms, Role in Physiology and Pathology (epmc).pdf DeepDyve Pubget Overpricing