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10.3389/fimmu.2020.585243 http://scihub22266oqcxt.onion/10.3389/fimmu.2020.585243 33193407!7609860!33193407     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=33193407&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       Front+Immunol 2020 ; 11 (?): 585243 Nephropedia Template TP {{tp|p=33193407|t=2020. The Influence of the Lectin Pathway of Complement Activation on Infections of the Respiratory System |pdf=|usr=}}{{33193407}} gab.com Text The Influence of the Lectin Pathway of Complement Activation on Infections of the Respiratory System JO: Front Immunol http://www.kidney.de/pget.php?&tw=1&pmid=33193407 #FOAvip Lung diseases are among the leading causes of morbidity and mortality. Complement activation may prevent a variety of respiratory infections, but on the other hand, could exacerbate tissue damage or contribute to adverse side effects. In this review, the associations of factors specific for complement activation via the lectin pathway (LP) with infections of the respiratory system, from birth to adulthood, are discussed. The most extensive data concern mannose-binding lectin (MBL) which together with other collectins (collectin-10, collectin-11) and the ficolins (ficolin-1, ficolin-2, ficolin-3) belong to pattern-recognition molecules (PRM) specific for the LP. Those PRM form complexes with MBL-associated serine proteases (MASP-1, MASP-2, MASP-3) and related non-enzymatic factors (MAp19, MAp44). Beside diseases affecting humanity for centuries like tuberculosis or neonatal pneumonia, some recently published data concerning COVID-19 are summarized. Twit Text FOAVip The Influence of the Lectin Pathway of Complement Activation on Infections of the Respiratory System ????Front Immunol http://www.kidney.de/pget.php?&tw=1&pmid=33193407 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=33193407 #FOAvip English Wikipedia <ref>{{Cite pmid|33193407}}</ref> The Influence of the Lectin Pathway of Complement Activation on Infections of the Respiratory System #MMPMID33193407 Swierzko AS; Cedzynski M Front Immunol 2020[]; 11 (?): 585243 PMID33193407show ga Lung diseases are among the leading causes of morbidity and mortality. Complement activation may prevent a variety of respiratory infections, but on the other hand, could exacerbate tissue damage or contribute to adverse side effects. In this review, the associations of factors specific for complement activation via the lectin pathway (LP) with infections of the respiratory system, from birth to adulthood, are discussed. The most extensive data concern mannose-binding lectin (MBL) which together with other collectins (collectin-10, collectin-11) and the ficolins (ficolin-1, ficolin-2, ficolin-3) belong to pattern-recognition molecules (PRM) specific for the LP. Those PRM form complexes with MBL-associated serine proteases (MASP-1, MASP-2, MASP-3) and related non-enzymatic factors (MAp19, MAp44). Beside diseases affecting humanity for centuries like tuberculosis or neonatal pneumonia, some recently published data concerning COVID-19 are summarized. |Animals[MESH] |COVID-19/genetics/*immunology/virology[MESH] |Complement Activation[MESH] |Complement Pathway, Mannose-Binding Lectin[MESH] |Complement System Proteins/genetics/*immunology[MESH] |Humans[MESH] |Mannose-Binding Lectin/genetics/*immunology[MESH] |Respiratory System/*immunology/virology[MESH]The Influence of the Lectin Pathway of Complement Activation on Infect(epmc).pdf DeepDyve Pubget Overpricing