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10.3390/ijms21218081

http://scihub22266oqcxt.onion/10.3390/ijms21218081
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33138181!7662604!33138181
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suck abstract from ncbi


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pmid33138181      Int+J+Mol+Sci 2020 ; 21 (21): ä
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  • Emerging Mechanisms of Pulmonary Vasoconstriction in SARS-CoV-2-Induced Acute Respiratory Distress Syndrome (ARDS) and Potential Therapeutic Targets #MMPMID33138181
  • Karmouty-Quintana H; Thandavarayan RA; Keller SP; Sahay S; Pandit LM; Akkanti B
  • Int J Mol Sci 2020[Oct]; 21 (21): ä PMID33138181show ga
  • The 1918 influenza killed approximately 50 million people in a few short years, and now, the world is facing another pandemic. In December 2019, a novel coronavirus named severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) has caused an international outbreak of a respiratory illness termed coronavirus disease 2019 (COVID-19) and rapidly spread to cause the worst pandemic since 1918. Recent clinical reports highlight an atypical presentation of acute respiratory distress syndrome (ARDS) in COVID-19 patients characterized by severe hypoxemia, an imbalance of the renin-angiotensin system, an increase in thrombogenic processes, and a cytokine release storm. These processes not only exacerbate lung injury but can also promote pulmonary vascular remodeling and vasoconstriction, which are hallmarks of pulmonary hypertension (PH). PH is a complication of ARDS that has received little attention; thus, we hypothesize that PH in COVID-19-induced ARDS represents an important target for disease amelioration. The mechanisms that can promote PH following SARS-CoV-2 infection are described. In this review article, we outline emerging mechanisms of pulmonary vascular dysfunction and outline potential treatment options that have been clinically tested.
  • |Acute Lung Injury/*pathology[MESH]
  • |Betacoronavirus[MESH]
  • |COVID-19[MESH]
  • |Coronavirus Infections/*drug therapy/*pathology[MESH]
  • |Cytokine Release Syndrome/drug therapy/pathology[MESH]
  • |Kallikrein-Kinin System/physiology[MESH]
  • |Pandemics[MESH]
  • |Pneumonia, Viral/*drug therapy/*pathology[MESH]
  • |Renin-Angiotensin System/physiology[MESH]
  • |SARS-CoV-2[MESH]
  • |Severe Acute Respiratory Syndrome/drug therapy/*pathology[MESH]


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