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10.1016/j.it.2020.10.004 http://scihub22266oqcxt.onion/10.1016/j.it.2020.10.004 33132005!7556779!33132005     free     free     free Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Trends+Immunol 2020 ; 41 (12): 1100-1115 Nephropedia Template TP {{tp|p=33132005|t=2020. Mechanisms of SARS-CoV-2 Transmission and Pathogenesis |pdf=|usr=}}{{33132005}} gab.com Text Mechanisms of SARS-CoV-2 Transmission and Pathogenesis JO: Trends Immunol http://www.kidney.de/pget.php?&tw=1&pmid=33132005 #FOAvip The emergence of severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) marks the third highly pathogenic coronavirus to spill over into the human population. SARS-CoV-2 is highly transmissible with a broad tissue tropism that is likely perpetuating the pandemic. However, important questions remain regarding its transmissibility and pathogenesis. In this review, we summarize current SARS-CoV-2 research, with an emphasis on transmission, tissue tropism, viral pathogenesis, and immune antagonism. We further present advances in animal models that are important for understanding the pathogenesis of SARS-CoV-2, vaccine development, and therapeutic testing. When necessary, comparisons are made from studies with SARS to provide further perspectives on coronavirus infectious disease 2019 (COVID-19), as well as draw inferences for future investigations. Twit Text FOAVip Mechanisms of SARS-CoV-2 Transmission and Pathogenesis ????Trends Immunol http://www.kidney.de/pget.php?&tw=1&pmid=33132005 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=33132005 #FOAvip English Wikipedia <ref>{{Cite pmid|33132005}}</ref> Mechanisms of SARS-CoV-2 Transmission and Pathogenesis #MMPMID33132005 Harrison AG; Lin T; Wang P Trends Immunol 2020[Dec]; 41 (12): 1100-1115 PMID33132005show ga The emergence of severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) marks the third highly pathogenic coronavirus to spill over into the human population. SARS-CoV-2 is highly transmissible with a broad tissue tropism that is likely perpetuating the pandemic. However, important questions remain regarding its transmissibility and pathogenesis. In this review, we summarize current SARS-CoV-2 research, with an emphasis on transmission, tissue tropism, viral pathogenesis, and immune antagonism. We further present advances in animal models that are important for understanding the pathogenesis of SARS-CoV-2, vaccine development, and therapeutic testing. When necessary, comparisons are made from studies with SARS to provide further perspectives on coronavirus infectious disease 2019 (COVID-19), as well as draw inferences for future investigations. |Animals[MESH] |COVID-19/*transmission[MESH] |Humans[MESH]Mechanisms of SARS-CoV-2 Transmission and Pathogenesis (epmc).pdf DeepDyve Pubget Overpricing