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10.1016/j.ijid.2020.10.059 http://scihub22266oqcxt.onion/10.1016/j.ijid.2020.10.059 33129966!7591873!33129966     free     free     free Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Int+J+Infect+Dis 2021 ; 102 (ä): 196-202 Nephropedia Template TP {{tp|p=33129966|t=2021. Could SARS-CoV-2-induced lung injury be attenuated by vitamin D?|pdf=|usr=}}{{33129966}} gab.com Text Could SARS-CoV-2-induced lung injury be attenuated by vitamin D JO: Int J Infect Dis http://www.kidney.de/pget.php?&tw=1&pmid=33129966 #FOAvip A novel coronavirus (severe acute respiratory syndrome coronavirus 2, SARS-CoV-2) has been confirmed as having the capacity to transmit from humans to humans, causing acute respiratory distress syndrome (ARDS) and acute lung injury. Angiotensin converting enzyme-2 (ACE2) is known to be expressed on type II pneumocytes. As a counter-regulatory arm of the renin-angiotensin system (RAS), ACE2 plays critical roles in the pathogenesis of ARDS and acute lung injury. The affinity of the spike protein receptor binding domain (RBD) of SARS-CoV-2 for human ACE2 (hACE2) largely determines the degree of clinical symptoms after infection by SARS-CoV-2. Previous studies have shown that regulating the ACE2/RAS system is effective in the treatment of severe acute respiratory syndrome coronavirus (SARS-CoV)-induced ARDS and acute lung injury. Since ACE2 is the host cell receptor for both SARS-CoV-2 and SARS-CoV, regulating the ACE2/RAS system may alleviate ARDS and acute lung injury caused by SARS-CoV-2 as well as SARS-CoV. Vitamin D was found to affect ACE2, the target of SARS-CoV-2; therefore, we propose that vitamin D might alleviate ARDS and acute lung injury induced by SARS-CoV-2 by modulating ACE2. Twit Text FOAVip Could SARS-CoV-2-induced lung injury be attenuated by vitamin D ????Int J Infect Dis http://www.kidney.de/pget.php?&tw=1&pmid=33129966 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=33129966 #FOAvip English Wikipedia <ref>{{Cite pmid|33129966}}</ref> Could SARS-CoV-2-induced lung injury be attenuated by vitamin D? #MMPMID33129966 Xiao D; Li X; Su X; Mu D; Qu Y Int J Infect Dis 2021[Jan]; 102 (ä): 196-202 PMID33129966show ga A novel coronavirus (severe acute respiratory syndrome coronavirus 2, SARS-CoV-2) has been confirmed as having the capacity to transmit from humans to humans, causing acute respiratory distress syndrome (ARDS) and acute lung injury. Angiotensin converting enzyme-2 (ACE2) is known to be expressed on type II pneumocytes. As a counter-regulatory arm of the renin-angiotensin system (RAS), ACE2 plays critical roles in the pathogenesis of ARDS and acute lung injury. The affinity of the spike protein receptor binding domain (RBD) of SARS-CoV-2 for human ACE2 (hACE2) largely determines the degree of clinical symptoms after infection by SARS-CoV-2. Previous studies have shown that regulating the ACE2/RAS system is effective in the treatment of severe acute respiratory syndrome coronavirus (SARS-CoV)-induced ARDS and acute lung injury. Since ACE2 is the host cell receptor for both SARS-CoV-2 and SARS-CoV, regulating the ACE2/RAS system may alleviate ARDS and acute lung injury caused by SARS-CoV-2 as well as SARS-CoV. Vitamin D was found to affect ACE2, the target of SARS-CoV-2; therefore, we propose that vitamin D might alleviate ARDS and acute lung injury induced by SARS-CoV-2 by modulating ACE2. |*SARS-CoV-2[MESH] |Acute Lung Injury/drug therapy/*etiology[MESH] |Angiotensin-Converting Enzyme 2/physiology[MESH] |COVID-19/*complications[MESH] |Humans[MESH] |Renin-Angiotensin System/physiology[MESH] |Respiratory Distress Syndrome/etiology[MESH]Could SARS-CoV-2-induced lung injury be attenuated by vitamin D?(epmc).pdf DeepDyve Pubget Overpricing