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10.1111/sji.12989

http://scihub22266oqcxt.onion/10.1111/sji.12989
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33113222!7645942!33113222
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suck abstract from ncbi

pmid33113222      Scand+J+Immunol 2021 ; 93 (3): e12989
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  • T cell immunobiology and cytokine storm of COVID-19 #MMPMID33113222
  • Luo XH; Zhu Y; Mao J; Du RC
  • Scand J Immunol 2021[Mar]; 93 (3): e12989 PMID33113222show ga
  • 2019 coronavirus disease (COVID-19) presents as a newly recognized pneumonia and could rapidly progress into acute respiratory distress syndrome which has brought about a global pandemic. Until now, no curative therapy has been strongly recommended for COVID-19 except for personalized supportive care. T cells and virus-specific T cells are essential to protect against virus infection, including COVID-19. Delayed immune reconstitution (IR) and cytokine storm (CS) remain serious obstacles for the cure of COVID-19. Most COVID-19 patients, especially among elderly patients, had marked lymphopenia and increased neutrophils, but T cell counts in severe COVID-19 patients surviving the disease gradually restored later. Elevated pro-inflammatory cytokines, particularly IL-6, IL-10, IL-2 and IL-17, and exhausted T cells are found in peripheral blood and the lungs. It suggests that Thymosin alpha1 and adoptive COVID-19-specific T cells could improve IR, while convalescent plasma, IL-6 blockade, mesenchymal stem cells and corticosteroids could suppress CS. More clinical studies in this field worldwide are urgently warranted to pave the way for therapy of COVID-19 in the future.
  • |COVID-19/epidemiology/*immunology/virology[MESH]
  • |Cytokine Release Syndrome/*immunology/metabolism[MESH]
  • |Cytokines/immunology/metabolism[MESH]
  • |Humans[MESH]
  • |Inflammation Mediators/immunology/metabolism[MESH]
  • |Lymphocyte Count[MESH]
  • |Pandemics/prevention & control[MESH]
  • |SARS-CoV-2/*immunology/physiology[MESH]
  • |T-Lymphocyte Subsets/immunology/metabolism[MESH]


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