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10.3389/fimmu.2020.580641 http://scihub22266oqcxt.onion/10.3389/fimmu.2020.580641 33101306!7554241!33101306     free     free     free       Front+Immunol 2020 ; 11 (ä): 580641 Nephropedia Template TP {{tp|p=33101306|t=2020. Covid-19: Perspectives on Innate Immune Evasion |pdf=|usr=}}{{33101306}} gab.com Text Covid-19: Perspectives on Innate Immune Evasion JO: Front Immunol http://www.kidney.de/pget.php?&tw=1&pmid=33101306 #FOAvip The ongoing outbreak of Coronavirus disease 2019 infection achieved pandemic status on March 11, 2020. As of September 8, 2020 it has caused over 890,000 mortalities world-wide. Coronaviral infections are enabled by potent immunoevasory mechanisms that target multiple aspects of innate immunity, with severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) able to induce a cytokine storm, impair interferon responses, and suppress antigen presentation on both MHC class I and class II. Understanding the immune responses to SARS-CoV-2 and its immunoevasion approaches will improve our understanding of pathogenesis, virus clearance, and contribute toward vaccine and immunotherepeutic design and evaluation. This review discusses the known host innate immune response and immune evasion mechanisms driving SARS-CoV-2 infection and pathophysiology. Twit Text FOAVip Covid-19: Perspectives on Innate Immune Evasion ????Front Immunol http://www.kidney.de/pget.php?&tw=1&pmid=33101306 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=33101306 #FOAvip English Wikipedia <ref>{{Cite pmid|33101306}}</ref> Covid-19: Perspectives on Innate Immune Evasion #MMPMID33101306 Taefehshokr N; Taefehshokr S; Hemmat N; Heit B Front Immunol 2020[]; 11 (ä): 580641 PMID33101306show ga The ongoing outbreak of Coronavirus disease 2019 infection achieved pandemic status on March 11, 2020. As of September 8, 2020 it has caused over 890,000 mortalities world-wide. Coronaviral infections are enabled by potent immunoevasory mechanisms that target multiple aspects of innate immunity, with severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) able to induce a cytokine storm, impair interferon responses, and suppress antigen presentation on both MHC class I and class II. Understanding the immune responses to SARS-CoV-2 and its immunoevasion approaches will improve our understanding of pathogenesis, virus clearance, and contribute toward vaccine and immunotherepeutic design and evaluation. This review discusses the known host innate immune response and immune evasion mechanisms driving SARS-CoV-2 infection and pathophysiology. |Betacoronavirus/*immunology[MESH] |COVID-19[MESH] |Coronavirus Infections/immunology/*pathology[MESH] |Cytokine Release Syndrome/immunology/pathology[MESH] |Cytokines/blood[MESH] |Humans[MESH] |Immune Evasion/*immunology[MESH] |Immunity, Innate/*immunology[MESH] |Pandemics[MESH] |Pneumonia, Viral/immunology/*pathology[MESH] |SARS-CoV-2[MESH]Covid-19: Perspectives on Innate Immune Evasion (epmc).pdf DeepDyve Pubget Overpricing