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10.1111/ene.14578

http://scihub22266oqcxt.onion/10.1111/ene.14578
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suck abstract from ncbi


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pmid33090599      Eur+J+Neurol 2021 ; 28 (10): 3503-3516
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  • Immunology of COVID-19 and disease-modifying therapies: The good, the bad and the unknown #MMPMID33090599
  • Zrzavy T; Wimmer I; Rommer PS; Berger T
  • Eur J Neurol 2021[Oct]; 28 (10): 3503-3516 PMID33090599show ga
  • OBJECTIVE: The outbreak of the SARS-CoV-2 pandemic, caused by a previously unknown infectious agent, posed unprecedented challenges to healthcare systems and unmasked their vulnerability and limitations worldwide. Patients with long-term immunomodulatory/suppressive therapies, as well as their physicians, were and are concerned about balancing the risk of infection and effects of disease-modifying therapy. Over the last few months, knowledge regarding SARS-CoV-2 has been growing tremendously, and the first experiences of infections in patients with multiple sclerosis (MS) have been reported. METHODS: This review summarizes the currently still limited knowledge about SARS-CoV-2 immunology and the commonly agreed modes of action of approved drugs in immune-mediated diseases of the central nervous system (MS and neuromyelitis optica spectrum disorder). Specifically, we discuss whether immunosuppressive/immunomodulatory drugs may increase the risk of SARS-CoV-2 infection and, conversely, may decrease the severity of a COVID-19 disease course. RESULTS: At present, it can be recommended in general that none of those therapies with a definite indication needs to be stopped per se. A possibly increased risk of infection for most medications is accompanied by the possibility to reduce the severity of COVID-19. CONCLUSIONS: Despite the knowledge gain over the last few months, current evidence remains limited, and, thus, further clinical vigilance and systematic documentation is essential.
  • |*COVID-19[MESH]
  • |*Multiple Sclerosis/drug therapy/epidemiology[MESH]
  • |*Neuromyelitis Optica/epidemiology[MESH]
  • |Humans[MESH]
  • |Pandemics[MESH]


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