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10.3390/jcm9103315

http://scihub22266oqcxt.onion/10.3390/jcm9103315
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suck abstract from ncbi


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pmid33076493      J+Clin+Med 2020 ; 9 (10): ä
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  • SARS-CoV-2 Viral Load, IFNlambda Polymorphisms and the Course of COVID-19: An Observational Study #MMPMID33076493
  • Amodio E; Pipitone RM; Grimaudo S; Immordino P; Maida CM; Prestileo T; Restivo V; Tramuto F; Vitale F; Craxi A; Casuccio A
  • J Clin Med 2020[Oct]; 9 (10): ä PMID33076493show ga
  • The course of SARS-CoV-2 infection ranges from asymptomatic to a multiorgan disease. In this observational study, we investigated SARS-CoV-2 infected subjects with defined outcomes, evaluating the relationship between viral load and single nucleotide polymorphisms of genes codifying for IFNlambdas (interferon). The study enrolled 381 patients with laboratory-confirmed SARS-CoV-2 infection. For each patient, a standardized form was filled including sociodemographic variables and clinical outcomes. The host's gene polymorphisms (IFNL3 rs1297860 C/T and INFL4 rs368234815 TT/DeltaG) and RtReal-Time PCR cycle threshold (PCR Ct) value on SARS-CoV-2 were assessed on nasal, pharyngeal or nasopharyngeal swabs. Higher viral loads were found in patients aged > 74 years and homozygous mutant polymorphisms DG in IFNL4 (adj-OR = 1.16, 95% CI = 1.01-1.34 and adj-OR = 1.24, 95% CI = 1.09-1.40, respectively). After adjusting for age and sex, a statistically significantly lower risk of hospitalization was observed in subjects with higher RtReal-Time PCR cycle threshold values (adj-OR = 0.95, 95% CI = 0.91, 0.99; p = 0.028). Our data support the correlation between SARS-CoV-2 load and disease severity, and suggest that IFNlambda polymorphisms could affect the ability of the host to modulate viral infection without a clear impact on the outcome of COVID-19.
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