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10.1007/s11239-020-02297-z http://scihub22266oqcxt.onion/10.1007/s11239-020-02297-z 33063256!7561230!33063256     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=33063256&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       J+Thromb+Thrombolysis 2021 ; 51 (3): 657-662 Nephropedia Template TP {{tp|p=33063256|t=2021. Coagulopathy, endothelial dysfunction, thrombotic microangiopathy and complement activation: potential role of complement system inhibition in COVID-19 |pdf=|usr=}}{{33063256}} gab.com Text Coagulopathy, endothelial dysfunction, thrombotic microangiopathy and complement activation: potential role of complement system inhibition in COVID-19 JO: J Thromb Thrombolysis http://www.kidney.de/pget.php?&tw=1&pmid=33063256 #FOAvip Coronavirus disease-2019 (COVID-19) is a rapidly evolving health crisis caused by the severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2). COVID-19 is a novel disease entity and we are in a learning phase with regards to the pathogenesis, disease manifestations, and therapeutics. In addition to the primary lung injury, many patients especially the ones with moderate to severe COVID-19 display evidence of endothelial damage, complement activation, which leads to a pro-coagulable state. While there are still missing links in our understanding, the interplay of endothelium, complement system activation, and immune response to the SARS-CoV-2 virus is a surprisingly major factor in COVID-19 pathogenesis. One could envision COVID-19 becoming a novel hematological syndrome. This review is to discuss the available literature with regards to the involvement of the complement system, and coagulation cascade and their interaction with endothelium. Twit Text FOAVip Coagulopathy, endothelial dysfunction, thrombotic microangiopathy and complement activation: potential role of complement system inhibition in COVID-19 ????J Thromb Thrombolysis http://www.kidney.de/pget.php?&tw=1&pmid=33063256 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=33063256 #FOAvip English Wikipedia <ref>{{Cite pmid|33063256}}</ref> Coagulopathy, endothelial dysfunction, thrombotic microangiopathy and complement activation: potential role of complement system inhibition in COVID-19 #MMPMID33063256 Wang X; Sahu KK; Cerny J J Thromb Thrombolysis 2021[Apr]; 51 (3): 657-662 PMID33063256show ga Coronavirus disease-2019 (COVID-19) is a rapidly evolving health crisis caused by the severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2). COVID-19 is a novel disease entity and we are in a learning phase with regards to the pathogenesis, disease manifestations, and therapeutics. In addition to the primary lung injury, many patients especially the ones with moderate to severe COVID-19 display evidence of endothelial damage, complement activation, which leads to a pro-coagulable state. While there are still missing links in our understanding, the interplay of endothelium, complement system activation, and immune response to the SARS-CoV-2 virus is a surprisingly major factor in COVID-19 pathogenesis. One could envision COVID-19 becoming a novel hematological syndrome. This review is to discuss the available literature with regards to the involvement of the complement system, and coagulation cascade and their interaction with endothelium. |*Complement Activation[MESH] |COVID-19/complications/immunology/*physiopathology/therapy[MESH] |Endothelium, Vascular/*physiopathology[MESH] |Humans[MESH] |Molecular Targeted Therapy[MESH]Coagulopathy, endothelial dysfunction, thrombotic microangiopathy and (epmc).pdf DeepDyve Pubget Overpricing