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10.1093/ajh/hpaa169 http://scihub22266oqcxt.onion/10.1093/ajh/hpaa169 33043967!7665331!33043967     free     free     free Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Am+J+Hypertens 2021 ; 34 (3): 278-281 Nephropedia Template TP {{tp|p=33043967|t=2021. Serum ACE2, Angiotensin II, and Aldosterone Levels Are Unchanged in Patients With COVID-19 |pdf=|usr=}}{{33043967}} gab.com Text Serum ACE2, Angiotensin II, and Aldosterone Levels Are Unchanged in Patients With COVID-19 JO: Am J Hypertens http://www.kidney.de/pget.php?&tw=1&pmid=33043967 #FOAvip BACKGROUND: The role of the renin-angiotensin-aldosterone system (RAAS) in coronavirus disease 2019 (COVID-19) is controversially discussed. Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) enters host cells by binding to angiotensin-converting enzyme 2 (ACE2) and activity of the RAAS may affect susceptibility to SARS-CoV-2 infection and outcome of patients with COVID-19. METHODS: In this prospective single-center study, we determined the serum levels of ACE2, angiotensin II, and aldosterone in patients with COVID-19 compared with control patients presenting with similar symptoms in the emergency unit. RESULTS: We analyzed serum samples from 24 SARS-CoV-2 positive and 61 SARS-CoV-2 negative patients. SARS-CoV-2 positive and control patients did not differ in baseline patients characteristics, symptoms, and clinical presentation. Mean serum concentrations of ACE2, angiotensin II, and aldosterone did not differ between the SARS-CoV-2 positive and the control group. In line with this, serum potassium as surrogate parameter for RAAS activity and blood pressure were similar in both groups. CONCLUSIONS: In summary, we did not find evidence for altered RAAS activity including angiotensin II, aldosterone, or potassium levels, and blood pressure in patients with COVID-19. CLINICAL TRIALS REGISTRATION: Trial Number DRKS00021206. Twit Text FOAVip Serum ACE2, Angiotensin II, and Aldosterone Levels Are Unchanged in Patients With COVID-19 ????Am J Hypertens http://www.kidney.de/pget.php?&tw=1&pmid=33043967 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=33043967 #FOAvip English Wikipedia <ref>{{Cite pmid|33043967}}</ref> Serum ACE2, Angiotensin II, and Aldosterone Levels Are Unchanged in Patients With COVID-19 #MMPMID33043967 Rieder M; Wirth L; Pollmeier L; Jeserich M; Goller I; Baldus N; Schmid B; Busch HJ; Hofmann M; Kern W; Bode C; Duerschmied D; Lother A Am J Hypertens 2021[Apr]; 34 (3): 278-281 PMID33043967show ga BACKGROUND: The role of the renin-angiotensin-aldosterone system (RAAS) in coronavirus disease 2019 (COVID-19) is controversially discussed. Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) enters host cells by binding to angiotensin-converting enzyme 2 (ACE2) and activity of the RAAS may affect susceptibility to SARS-CoV-2 infection and outcome of patients with COVID-19. METHODS: In this prospective single-center study, we determined the serum levels of ACE2, angiotensin II, and aldosterone in patients with COVID-19 compared with control patients presenting with similar symptoms in the emergency unit. RESULTS: We analyzed serum samples from 24 SARS-CoV-2 positive and 61 SARS-CoV-2 negative patients. SARS-CoV-2 positive and control patients did not differ in baseline patients characteristics, symptoms, and clinical presentation. Mean serum concentrations of ACE2, angiotensin II, and aldosterone did not differ between the SARS-CoV-2 positive and the control group. In line with this, serum potassium as surrogate parameter for RAAS activity and blood pressure were similar in both groups. CONCLUSIONS: In summary, we did not find evidence for altered RAAS activity including angiotensin II, aldosterone, or potassium levels, and blood pressure in patients with COVID-19. CLINICAL TRIALS REGISTRATION: Trial Number DRKS00021206. |*COVID-19/blood/diagnosis/epidemiology/physiopathology[MESH] |*Hypertension/blood/drug therapy/epidemiology/physiopathology[MESH] |Aldosterone/*blood[MESH] |Angiotensin II/*blood[MESH] |Angiotensin-Converting Enzyme 2/*blood[MESH] |Angiotensin-Converting Enzyme Inhibitors/therapeutic use[MESH] |Blood Pressure Determination/statistics & numerical data[MESH] |Female[MESH] |Germany/epidemiology[MESH] |Humans[MESH] |Male[MESH] |Middle Aged[MESH] |Outcome Assessment, Health Care[MESH] |Potassium/*blood[MESH] |Prospective Studies[MESH] |Renin-Angiotensin System/physiology[MESH]Serum ACE2, Angiotensin II, and Aldosterone Levels Are Unchanged in Pa(epmc).pdf DeepDyve Pubget Overpricing