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10.1172/JCI143361 http://scihub22266oqcxt.onion/10.1172/JCI143361 33021506!7685713!33021506     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       J+Clin+Invest 2020 ; 130 (12): 6214-6217 Nephropedia Template TP {{tp|p=33021506|t=2020. COVID-19 and myeloid cells: complex interplay correlates with lung severity |pdf=|usr=}}{{33021506}} gab.com Text COVID-19 and myeloid cells: complex interplay correlates with lung severity JO: J Clin Invest http://www.kidney.de/pget.php?&tw=1&pmid=33021506 #FOAvip COVID-19 spans a wide range of symptoms, sometimes with profound immune system involvement. How immune cell subsets change during the disease course and with disease severity needs further study. While myeloid cells have been shown to initiate and maintain responses to pneumonia and lung inflammation, often playing a role in resolution, their involvement with COVID-19 remains unknown. In this issue of the JCI, Sanchez-Cerrillo and Landete et al. investigated DCs and monocytes from blood and bronchial secretions of patients with varying COVID-19 severity and with healthy controls. The authors conclude that circulating monocytes and DCs migrate from the blood into the inflamed lungs. While sampling differences in sex, collection timing, bacteria/fungal infection, and corticosteroid treatment limit interpretation, we believe that reprogramming monocyte or macrophages by targeting immunometabolism, epigenetics, or the cytokine milieu holds promise in resolving lung inflammation associated with COVID-19. Twit Text FOAVip COVID-19 and myeloid cells: complex interplay correlates with lung severity ????J Clin Invest http://www.kidney.de/pget.php?&tw=1&pmid=33021506 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=33021506 #FOAvip English Wikipedia <ref>{{Cite pmid|33021506}}</ref> COVID-19 and myeloid cells: complex interplay correlates with lung severity #MMPMID33021506 D'Alessio FR; Heller NM J Clin Invest 2020[Dec]; 130 (12): 6214-6217 PMID33021506show ga COVID-19 spans a wide range of symptoms, sometimes with profound immune system involvement. How immune cell subsets change during the disease course and with disease severity needs further study. While myeloid cells have been shown to initiate and maintain responses to pneumonia and lung inflammation, often playing a role in resolution, their involvement with COVID-19 remains unknown. In this issue of the JCI, Sanchez-Cerrillo and Landete et al. investigated DCs and monocytes from blood and bronchial secretions of patients with varying COVID-19 severity and with healthy controls. The authors conclude that circulating monocytes and DCs migrate from the blood into the inflamed lungs. While sampling differences in sex, collection timing, bacteria/fungal infection, and corticosteroid treatment limit interpretation, we believe that reprogramming monocyte or macrophages by targeting immunometabolism, epigenetics, or the cytokine milieu holds promise in resolving lung inflammation associated with COVID-19. |*COVID-19[MESH] |Humans[MESH] |Lung[MESH] |Monocytes[MESH] |Pandemics[MESH]COVID-19 and myeloid cells: complex interplay correlates with lung sev(epmc).pdf DeepDyve Pubget Overpricing