Use my Search Websuite to scan PubMed, PMCentral, Journal Hosts and Journal Archives, FullText. Kick-your-searchterm to multiple Engines kick-your-query now !>

A dictionary by aggregated review articles of nephrology, medicine and the life sciences Your one-stop-run pathway from word to the immediate pdf of peer-reviewed on-topic knowledge.

10.1016/j.mehy.2020.110201 http://scihub22266oqcxt.onion/10.1016/j.mehy.2020.110201 33017909!7430244!33017909     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=33017909&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       Med+Hypotheses 2020 ; 143 (?): 110201 Nephropedia Template TP {{tp|p=33017909|t=2020. Could aquaporin modulators be employed as prospective drugs for COVID-19 related pulmonary comorbidity?|pdf=|usr=}}{{33017909}} gab.com Text Could aquaporin modulators be employed as prospective drugs for COVID-19 related pulmonary comorbidity JO: Med Hypotheses http://www.kidney.de/pget.php?&tw=1&pmid=33017909 #FOAvip COVID-19 initially an epidemic caused by SARS-CoV-2 has turned out to be a life- threatening global pandemic with increased morbidity and mortality. The presence of cytokine storm has been linked with the pathogenesis of severe lung injury as evinced in COVID-19. Aquaporins (AQPs) are molecular water channels, facilitating water transport across the cell membrane in response to osmotic gradients. Impairment in alveolar fluid clearance due to altered functional expression of respiratory AQPs highlight their pathophysiological significance in pulmonary edema associated respiratory illness. Therefore, we hypothesize that targeted modulation of AQPs in lungs in the intervening period of time, could diminish the dreadful effects of inflammation- induced comorbidity in COVID-19. Twit Text FOAVip Could aquaporin modulators be employed as prospective drugs for COVID-19 related pulmonary comorbidity ????Med Hypotheses http://www.kidney.de/pget.php?&tw=1&pmid=33017909 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=33017909 #FOAvip English Wikipedia <ref>{{Cite pmid|33017909}}</ref> Could aquaporin modulators be employed as prospective drugs for COVID-19 related pulmonary comorbidity? #MMPMID33017909 Mariajoseph-Antony LF; Kannan A; Panneerselvam A; Loganathan C; Anbarasu K; Prahalathan C Med Hypotheses 2020[Oct]; 143 (?): 110201 PMID33017909show ga COVID-19 initially an epidemic caused by SARS-CoV-2 has turned out to be a life- threatening global pandemic with increased morbidity and mortality. The presence of cytokine storm has been linked with the pathogenesis of severe lung injury as evinced in COVID-19. Aquaporins (AQPs) are molecular water channels, facilitating water transport across the cell membrane in response to osmotic gradients. Impairment in alveolar fluid clearance due to altered functional expression of respiratory AQPs highlight their pathophysiological significance in pulmonary edema associated respiratory illness. Therefore, we hypothesize that targeted modulation of AQPs in lungs in the intervening period of time, could diminish the dreadful effects of inflammation- induced comorbidity in COVID-19. |Animals[MESH] |Aquaporins/*metabolism[MESH] |Betacoronavirus[MESH] |Biological Transport[MESH] |COVID-19[MESH] |COVID-19 Drug Treatment[MESH] |Comorbidity[MESH] |Coronavirus Infections/*drug therapy[MESH] |Cytokines/metabolism[MESH] |Humans[MESH] |Inflammation[MESH] |Lung/immunology/virology[MESH] |Mice[MESH] |Pandemics[MESH] |Pneumonia, Viral/*drug therapy[MESH] |Pulmonary Edema/*drug therapy[MESH]Could aquaporin modulators be employed as prospective drugs for COVID-(epmc).pdf DeepDyve Pubget Overpricing