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10.1016/j.isci.2020.101631 http://scihub22266oqcxt.onion/10.1016/j.isci.2020.101631 33015593!7524535!33015593     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\33015593.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       iScience 2020 ; 23 (10): 101631 Nephropedia Template TP {{tp|p=33015593|t=2020. Mitochondria: In the Cross Fire of SARS-CoV-2 and Immunity |pdf=|usr=}}{{33015593}} gab.com Text Mitochondria: In the Cross Fire of SARS-CoV-2 and Immunity JO: iScience http://www.kidney.de/pget.php?&tw=1&pmid=33015593 #FOAvip The pathophysiology, immune reaction, and differential vulnerability of different population groups and viral host immune system evasion strategies of severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infection are not yet well understood. Here, we reviewed the multitude of known strategies of coronaviruses and other viruses to usurp mitochondria-associated mechanisms involved in the host innate immune response and put them in context with the current knowledge on SARS-CoV-2. We argue that maintenance of mitochondrial integrity is essential for adequate innate immune system responses and to blunt mitochondrial modulation by SARS-CoV-2. Mitochondrial health thus may determine differential vulnerabilities to SARS-CoV-2 infection rendering markers of mitochondrial functions promising potential biomarkers for SARS-CoV-2 infection risk and severity of outcome. Current knowledge gaps on our understanding of mitochondrial involvement in SARS-CoV-2 infection, lifestyle, and pharmacological strategies to improve mitochondrial integrity and potential reciprocal interactions with chronic and age-related diseases, e.g., Parkinson disease, are pointed out. Twit Text FOAVip Mitochondria: In the Cross Fire of SARS-CoV-2 and Immunity ????iScience http://www.kidney.de/pget.php?&tw=1&pmid=33015593 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=33015593 #FOAvip English Wikipedia <ref>{{Cite pmid|33015593}}</ref> Mitochondria: In the Cross Fire of SARS-CoV-2 and Immunity #MMPMID33015593 Burtscher J; Cappellano G; Omori A; Koshiba T; Millet GP iScience 2020[Oct]; 23 (10): 101631 PMID33015593show ga The pathophysiology, immune reaction, and differential vulnerability of different population groups and viral host immune system evasion strategies of severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infection are not yet well understood. Here, we reviewed the multitude of known strategies of coronaviruses and other viruses to usurp mitochondria-associated mechanisms involved in the host innate immune response and put them in context with the current knowledge on SARS-CoV-2. We argue that maintenance of mitochondrial integrity is essential for adequate innate immune system responses and to blunt mitochondrial modulation by SARS-CoV-2. Mitochondrial health thus may determine differential vulnerabilities to SARS-CoV-2 infection rendering markers of mitochondrial functions promising potential biomarkers for SARS-CoV-2 infection risk and severity of outcome. Current knowledge gaps on our understanding of mitochondrial involvement in SARS-CoV-2 infection, lifestyle, and pharmacological strategies to improve mitochondrial integrity and potential reciprocal interactions with chronic and age-related diseases, e.g., Parkinson disease, are pointed out. äMitochondria: In the Cross Fire of SARS-CoV-2 and Immunity (epmc).pdf DeepDyve Pubget Overpricing