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10.1016/j.virusres.2020.198176

http://scihub22266oqcxt.onion/10.1016/j.virusres.2020.198176
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32987033!7518800!32987033
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suck abstract from ncbi

pmid32987033      Virus+Res 2020 ; 290 (ä): 198176
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  • Transcription-based drug repurposing for COVID-19 #MMPMID32987033
  • Killick R; Ballard C; Doherty P; Williams G
  • Virus Res 2020[Dec]; 290 (ä): 198176 PMID32987033show ga
  • We have utilised the transcriptional response of lung epithelial cells following infection by the original Severe Acute Respiratory Syndrome coronavirus (SARS) to identify repurposable drugs for COVID-19. Drugs best able to recapitulate the infection profile are highly enriched for antiviral activity. Nine of these have been tested against SARS-2 and found to potently antagonise SARS-2 infection/replication, with a number now being considered for clinical trials. It is hoped that this approach may serve to broaden the spectrum of approved drugs that should be further assessed as potential anti-COVID-19 agents and may help elucidate how this seemingly disparate collection of drugs are able to inhibit SARS-2 infection/replication.
  • |*COVID-19 Drug Treatment[MESH]
  • |*Drug Repositioning[MESH]
  • |Antiviral Agents/chemistry/*pharmacology/therapeutic use[MESH]
  • |COVID-19/virology[MESH]
  • |Epithelial Cells/drug effects/pathology/virology[MESH]
  • |Gene Expression Profiling[MESH]
  • |Humans[MESH]
  • |Lung/drug effects/pathology/virology[MESH]
  • |SARS-CoV-2/*drug effects/physiology[MESH]
  • |Severe acute respiratory syndrome-related coronavirus/drug effects/physiology[MESH]


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