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10.1111/iwj.13483 http://scihub22266oqcxt.onion/10.1111/iwj.13483 32986928!7536990!32986928     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=32986928&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Deprecated: Implicit conversion from float 215.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 215.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 215.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Int+Wound+J 2020 ; 17 (6): 1935-1940 Nephropedia Template TP {{tp|p=32986928|t=2020. Pathophysiology of coronavirus disease 2019 for wound care professionals |pdf=|usr=}}{{32986928}} gab.com Text Pathophysiology of coronavirus disease 2019 for wound care professionals JO: Int Wound J http://www.kidney.de/pget.php?&tw=1&pmid=32986928 #FOAvip There is pressing urgency to understand the pathogenesis of the severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) which causes Coronavirus disease 2019 (COVID-19). The tissue tropism of SARS-CoV-2 includes not only the lung but also the vascular and integumentary systems. Angiotensin-converting enzyme 2 (ACE2) appears to be the key functional receptor for the virus. There is a prominent innate immune response to SARS-CoV-2 infection, including inflammatory cytokines, chemokines, the complement system, and acute phase proteins. The pathophysiologic significance of SARS-COV-2 and host immune system interaction, and COVID-19-associated coagulopathy instigating microvascular injury syndrome mediated by activation of complement pathways, and an associated procoagulant state is important for wound care professionals to understand. Twit Text FOAVip Pathophysiology of coronavirus disease 2019 for wound care professionals ????Int Wound J http://www.kidney.de/pget.php?&tw=1&pmid=32986928 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=32986928 #FOAvip English Wikipedia <ref>{{Cite pmid|32986928}}</ref> Pathophysiology of coronavirus disease 2019 for wound care professionals #MMPMID32986928 Al-Benna S Int Wound J 2020[Dec]; 17 (6): 1935-1940 PMID32986928show ga There is pressing urgency to understand the pathogenesis of the severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) which causes Coronavirus disease 2019 (COVID-19). The tissue tropism of SARS-CoV-2 includes not only the lung but also the vascular and integumentary systems. Angiotensin-converting enzyme 2 (ACE2) appears to be the key functional receptor for the virus. There is a prominent innate immune response to SARS-CoV-2 infection, including inflammatory cytokines, chemokines, the complement system, and acute phase proteins. The pathophysiologic significance of SARS-COV-2 and host immune system interaction, and COVID-19-associated coagulopathy instigating microvascular injury syndrome mediated by activation of complement pathways, and an associated procoagulant state is important for wound care professionals to understand. |*Health Personnel[MESH] |*Immunity, Innate[MESH] |COVID-19/*epidemiology/immunology[MESH] |Comorbidity[MESH] |Humans[MESH] |SARS-CoV-2[MESH]Pathophysiology of coronavirus disease 2019 for wound care professiona(epmc).pdf DeepDyve Pubget Overpricing