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10.2147/JIR.S267280

http://scihub22266oqcxt.onion/10.2147/JIR.S267280
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32982366!7490109!32982366
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suck abstract from ncbi


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pmid32982366      J+Inflamm+Res 2020 ; 13 (ä): 507-518
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  • Role of IFN and Complements System: Innate Immunity in SARS-CoV-2 #MMPMID32982366
  • Shibabaw T; Molla MD; Teferi B; Ayelign B
  • J Inflamm Res 2020[]; 13 (ä): 507-518 PMID32982366show ga
  • The critical role of the innate immune system has been confirmed in driving local and systemic inflammation and the cytokine release storm in severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2). This dysregulated immune response is focused on interferon (IFN) and complement activation, which are crucial for the development of metabolic inflammation, local lung tissue damage, and systemic multi-organ failure. IFNs control viral infections by inducing expression of IFN-stimulated genes (ISGs) that restrict distinct steps of viral replication. Therefore, in this review article, we propose the mechanism of SARS-CoV-2-associated acute respiratory disease syndrome, and assess treatment options by considering IFNs and by targeting IFN-antagonist SARS-CoV-2 virulent gene products. Furthermore, we elaborate on the mechanism of the amplified complement-mediated inflammatory cytokine storm, and propose an antiviral and immunotherapeutic strategy against coronavirus disease 2019 (COVID-19).
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