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10.3349/ymj.2020.61.10.891

http://scihub22266oqcxt.onion/10.3349/ymj.2020.61.10.891
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suck abstract from ncbi


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pmid32975064      Yonsei+Med+J 2020 ; 61 (10): 891-894
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  • Quantitative Proteomic Analysis of the Expression of SARS-CoV-2 Receptors in the Gut of Patients with Chronic Enterocolitis #MMPMID32975064
  • Park J; Jeong D; Chung YW; Kim DH; Cheon JH; Ryu JH
  • Yonsei Med J 2020[Oct]; 61 (10): 891-894 PMID32975064show ga
  • The cellular entry of severe respiratory syndrome coronavirus-2 (SARS-CoV-2) is mediated by interaction with the human angiotensin-converting enzyme 2 (ACE2), a receptor that is expressed on both lung and intestinal epithelial cells. We performed a quantitative proteomic analysis to investigate the expression of possible receptors for SARS-CoV-2 in the intestinal mucosa of 23 patients with chronic colitis. ACE2 expression was low and remained unaltered in the gut of patients with ulcerative colitis (UC), Crohn's disease (CD), intestinal Behcet's disease (BD), and intestinal tuberculosis (TB), when compared with that of healthy individuals. Additionally, the expression levels of some probable co-receptors, including dipeptidyl peptidase 4 (DPP4), aminopeptidase N (AMPN), and glutamyl aminopeptidase (AMPE), were unchanged in the affected UC, CD, intestinal BD, and intestinal TB colon mucosa samples. In conclusion, gut inflammation associated with chronic colitis does not mediate a further increase in the cellular entry of SARS-CoV-2.
  • |*Coronavirus Infections[MESH]
  • |*Enterocolitis/metabolism[MESH]
  • |*Pneumonia, Viral[MESH]
  • |*Proteomics[MESH]
  • |Angiotensin-Converting Enzyme 2[MESH]
  • |Betacoronavirus[MESH]
  • |COVID-19[MESH]
  • |Dipeptidyl Peptidase 4/metabolism[MESH]
  • |Gastrointestinal Microbiome[MESH]
  • |Humans[MESH]
  • |Lung/metabolism[MESH]
  • |Pandemics[MESH]
  • |Peptidyl-Dipeptidase A/*metabolism[MESH]


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