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10.1016/j.jacep.2020.08.002 http://scihub22266oqcxt.onion/10.1016/j.jacep.2020.08.002 32972561!7417167!32972561     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=32972561&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       JACC+Clin+Electrophysiol 2020 ; 6 (9): 1193-1204 Nephropedia Template TP {{tp|p=32972561|t=2020. Arrhythmias and COVID-19: A Review |pdf=|usr=}}{{32972561}} gab.com Text Arrhythmias and COVID-19: A Review JO: JACC Clin Electrophysiol http://www.kidney.de/pget.php?&tw=1&pmid=32972561 #FOAvip Current understanding of the impact of coronavirus disease-2019 (COVID-19) on arrhythmias continues to evolve as new data emerge. Cardiac arrhythmias are more common in critically ill COVID-19 patients. The potential mechanisms that could result in arrhythmogenesis among COVID-19 patients include hypoxia caused by direct viral tissue involvement of lungs, myocarditis, abnormal host immune response, myocardial ischemia, myocardial strain, electrolyte derangements, intravascular volume imbalances, and drug sides effects. To manage these arrhythmias, it is imperative to increase the awareness of potential drug-drug interactions, to monitor QTc prolongation while receiving COVID therapy and provide special considerations for patients with inherited arrhythmia syndromes. It is also crucial to minimize exposure to COVID-19 infection by stratifying the need for intervention and using telemedicine. As COVID-19 infection continues to prevail with a potential for future surges, more data are required to better understand pathophysiology and to validate management strategies. Twit Text FOAVip Arrhythmias and COVID-19: A Review ????JACC Clin Electrophysiol http://www.kidney.de/pget.php?&tw=1&pmid=32972561 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=32972561 #FOAvip English Wikipedia <ref>{{Cite pmid|32972561}}</ref> Arrhythmias and COVID-19: A Review #MMPMID32972561 Dherange P; Lang J; Qian P; Oberfeld B; Sauer WH; Koplan B; Tedrow U JACC Clin Electrophysiol 2020[Sep]; 6 (9): 1193-1204 PMID32972561show ga Current understanding of the impact of coronavirus disease-2019 (COVID-19) on arrhythmias continues to evolve as new data emerge. Cardiac arrhythmias are more common in critically ill COVID-19 patients. The potential mechanisms that could result in arrhythmogenesis among COVID-19 patients include hypoxia caused by direct viral tissue involvement of lungs, myocarditis, abnormal host immune response, myocardial ischemia, myocardial strain, electrolyte derangements, intravascular volume imbalances, and drug sides effects. To manage these arrhythmias, it is imperative to increase the awareness of potential drug-drug interactions, to monitor QTc prolongation while receiving COVID therapy and provide special considerations for patients with inherited arrhythmia syndromes. It is also crucial to minimize exposure to COVID-19 infection by stratifying the need for intervention and using telemedicine. As COVID-19 infection continues to prevail with a potential for future surges, more data are required to better understand pathophysiology and to validate management strategies. |*Betacoronavirus[MESH] |*Pandemics[MESH] |Arrhythmias, Cardiac/epidemiology/*etiology[MESH] |COVID-19[MESH] |Coronavirus Infections/*complications/epidemiology[MESH] |Global Health[MESH] |Humans[MESH] |Morbidity/trends[MESH] |Pneumonia, Viral/*complications/epidemiology[MESH]Arrhythmias and COVID-19: A Review (epmc).pdf DeepDyve Pubget Overpricing