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10.1016/j.cell.2020.09.016 http://scihub22266oqcxt.onion/10.1016/j.cell.2020.09.016 32966765!7474869!32966765     free     free     free Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Cell 2020 ; 183 (4): 968-981.e7 Nephropedia Template TP {{tp|p=32966765|t=2020. The Immunology of Multisystem Inflammatory Syndrome in Children with COVID-19 |pdf=|usr=}}{{32966765}} gab.com Text The Immunology of Multisystem Inflammatory Syndrome in Children with COVID-19 JO: Cell http://www.kidney.de/pget.php?&tw=1&pmid=32966765 #FOAvip Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infection is typically very mild and often asymptomatic in children. A complication is the rare multisystem inflammatory syndrome in children (MIS-C) associated with COVID-19, presenting 4-6 weeks after infection as high fever, organ dysfunction, and strongly elevated markers of inflammation. The pathogenesis is unclear but has overlapping features with Kawasaki disease suggestive of vasculitis and a likely autoimmune etiology. We apply systems-level analyses of blood immune cells, cytokines, and autoantibodies in healthy children, children with Kawasaki disease enrolled prior to COVID-19, children infected with SARS-CoV-2, and children presenting with MIS-C. We find that the inflammatory response in MIS-C differs from the cytokine storm of severe acute COVID-19, shares several features with Kawasaki disease, but also differs from this condition with respect to T cell subsets, interleukin (IL)-17A, and biomarkers associated with arterial damage. Finally, autoantibody profiling suggests multiple autoantibodies that could be involved in the pathogenesis of MIS-C. Twit Text FOAVip The Immunology of Multisystem Inflammatory Syndrome in Children with COVID-19 ????Cell http://www.kidney.de/pget.php?&tw=1&pmid=32966765 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=32966765 #FOAvip English Wikipedia <ref>{{Cite pmid|32966765}}</ref> The Immunology of Multisystem Inflammatory Syndrome in Children with COVID-19 #MMPMID32966765 Consiglio CR; Cotugno N; Sardh F; Pou C; Amodio D; Rodriguez L; Tan Z; Zicari S; Ruggiero A; Pascucci GR; Santilli V; Campbell T; Bryceson Y; Eriksson D; Wang J; Marchesi A; Lakshmikanth T; Campana A; Villani A; Rossi P; Landegren N; Palma P; Brodin P Cell 2020[Nov]; 183 (4): 968-981.e7 PMID32966765show ga Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infection is typically very mild and often asymptomatic in children. A complication is the rare multisystem inflammatory syndrome in children (MIS-C) associated with COVID-19, presenting 4-6 weeks after infection as high fever, organ dysfunction, and strongly elevated markers of inflammation. The pathogenesis is unclear but has overlapping features with Kawasaki disease suggestive of vasculitis and a likely autoimmune etiology. We apply systems-level analyses of blood immune cells, cytokines, and autoantibodies in healthy children, children with Kawasaki disease enrolled prior to COVID-19, children infected with SARS-CoV-2, and children presenting with MIS-C. We find that the inflammatory response in MIS-C differs from the cytokine storm of severe acute COVID-19, shares several features with Kawasaki disease, but also differs from this condition with respect to T cell subsets, interleukin (IL)-17A, and biomarkers associated with arterial damage. Finally, autoantibody profiling suggests multiple autoantibodies that could be involved in the pathogenesis of MIS-C. |Autoantibodies/blood[MESH] |Betacoronavirus/isolation & purification[MESH] |COVID-19[MESH] |Child[MESH] |Child, Preschool[MESH] |Coronavirus Infections/complications/*pathology/virology[MESH] |Cytokines/metabolism[MESH] |Female[MESH] |Humans[MESH] |Immunity, Humoral[MESH] |Infant[MESH] |Male[MESH] |Mucocutaneous Lymph Node Syndrome/complications/immunology/pathology[MESH] |Pandemics[MESH] |Pneumonia, Viral/complications/*pathology/virology[MESH] |Principal Component Analysis[MESH] |Proteome/analysis[MESH] |SARS-CoV-2[MESH] |Severity of Illness Index[MESH] |Systemic Inflammatory Response Syndrome/etiology/immunology/*pathology[MESH]The Immunology of Multisystem Inflammatory Syndrome in Children with C(epmc).pdf DeepDyve Pubget Overpricing