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10.1016/j.resp.2020.103548 http://scihub22266oqcxt.onion/10.1016/j.resp.2020.103548 32956843!7500408!32956843     free     free     free Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Respir+Physiol+Neurobiol 2021 ; 283 (ä): 103548 Nephropedia Template TP {{tp|p=32956843|t=2021. Angiotensin-converting enzyme 2 (ACE2): COVID 19 gate way to multiple organ failure syndromes |pdf=|usr=}}{{32956843}} gab.com Text Angiotensin-converting enzyme 2 (ACE2): COVID 19 gate way to multiple organ failure syndromes JO: Respir Physiol Neurobiol http://www.kidney.de/pget.php?&tw=1&pmid=32956843 #FOAvip BACKGROUND: Globally, the current medical emergency for novel coronavirus 2019 (COVID-19) leads to respiratory distress syndrome and death. PURPOSE: This review highlighted the effect of COVID-19 on systemic multiple organ failure syndromes. This review is intended to fill a gap in information about human physiological response to COVID-19 infections. This review may shed some light on other potential mechanisms and approaches in COVID -19 infections towards systemic multiorgan failure syndromes. FINDING: SARS-CoV-2 intervened mainly in the lung with progression to pneumonia and acute respiratory distress syndrome (ARDS) via the angiotensin-converting enzyme 2(ACE2) receptor. Depending on the viral load, infection spread through the ACE2 receptor further to various organs such as heart, liver, kidney, brain, endothelium, GIT, immune cell, and RBC (thromboembolism). This may be aggravated by cytokine storm with the extensive release of proinflammatory cytokines from the deregulating immune system. CONCLUSION: The widespread and vicious combinations of cytokines with organ crosstalk contribute to systemic hyper inflammation and ultimately lead to multiple organ dysfunction (Fig. 1). This comprehensive study comprises various manifestations of different organs in COVID-19 and may assist the clinicians and scientists pertaining to a broad approach to fight COVID 19. Twit Text FOAVip Angiotensin-converting enzyme 2 (ACE2): COVID 19 gate way to multiple organ failure syndromes ????Respir Physiol Neurobiol http://www.kidney.de/pget.php?&tw=1&pmid=32956843 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=32956843 #FOAvip English Wikipedia <ref>{{Cite pmid|32956843}}</ref> Angiotensin-converting enzyme 2 (ACE2): COVID 19 gate way to multiple organ failure syndromes #MMPMID32956843 Loganathan S; Kuppusamy M; Wankhar W; Gurugubelli KR; Mahadevappa VH; Lepcha L; Choudhary AK Respir Physiol Neurobiol 2021[Jan]; 283 (ä): 103548 PMID32956843show ga BACKGROUND: Globally, the current medical emergency for novel coronavirus 2019 (COVID-19) leads to respiratory distress syndrome and death. PURPOSE: This review highlighted the effect of COVID-19 on systemic multiple organ failure syndromes. This review is intended to fill a gap in information about human physiological response to COVID-19 infections. This review may shed some light on other potential mechanisms and approaches in COVID -19 infections towards systemic multiorgan failure syndromes. FINDING: SARS-CoV-2 intervened mainly in the lung with progression to pneumonia and acute respiratory distress syndrome (ARDS) via the angiotensin-converting enzyme 2(ACE2) receptor. Depending on the viral load, infection spread through the ACE2 receptor further to various organs such as heart, liver, kidney, brain, endothelium, GIT, immune cell, and RBC (thromboembolism). This may be aggravated by cytokine storm with the extensive release of proinflammatory cytokines from the deregulating immune system. CONCLUSION: The widespread and vicious combinations of cytokines with organ crosstalk contribute to systemic hyper inflammation and ultimately lead to multiple organ dysfunction (Fig. 1). This comprehensive study comprises various manifestations of different organs in COVID-19 and may assist the clinicians and scientists pertaining to a broad approach to fight COVID 19. |Acute Kidney Injury/immunology/physiopathology[MESH] |Angiotensin-Converting Enzyme 2[MESH] |Arrhythmias, Cardiac/immunology/physiopathology[MESH] |Betacoronavirus/metabolism[MESH] |COVID-19[MESH] |Coronavirus Infections/*immunology/physiopathology[MESH] |Cytokine Release Syndrome/*immunology/physiopathology[MESH] |Cytokines/immunology[MESH] |Endothelium, Vascular/metabolism[MESH] |Erythrocytes/metabolism[MESH] |Gastrointestinal Diseases/immunology/physiopathology[MESH] |Gastrointestinal Tract/metabolism[MESH] |Heart Failure/immunology/physiopathology[MESH] |Humans[MESH] |Inflammation/immunology[MESH] |Kidney/metabolism[MESH] |Liver Diseases/immunology/physiopathology[MESH] |Liver/metabolism[MESH] |Lung/metabolism[MESH] |Multiple Organ Failure/*immunology/physiopathology[MESH] |Myocardium/metabolism[MESH] |Pandemics[MESH] |Peptidyl-Dipeptidase A/*metabolism[MESH] |Pneumonia, Viral/*immunology/physiopathology[MESH] |Respiratory Distress Syndrome/*immunology/physiopathology[MESH] |SARS-CoV-2[MESH] |Spike Glycoprotein, Coronavirus/*metabolism[MESH] |Thromboembolism/immunology/physiopathology[MESH]Angiotensin-converting enzyme 2 (ACE2): COVID 19 gate way to multiple (epmc).pdf DeepDyve Pubget Overpricing